Category: Uncategorized

Throat Mist

Instant Help for Sore Throats!

Throat Mist doesn’t just “numb” a sore throat, it gives instant relief but also contains herbs which stimulate the immune system.

Contains: Echinacea Royal, propolis, Ligusticum (Osha), Hyssop, Prickly ash bark and essential oil of orange.

Use: Spray in back of throat on inflamed tissues. As needed up to every hour.

Click here to
Order This Concierge Product WellevateThis is a separate website and you will need to create a new account to order.

Longevity & Rejuvenation

Live A Long and Healthy Life

Many of the practices that make for a healthier life also increase life expectancy. In addition, some herbs and nutritional substances may increase life expectancy, although this is less well-proven than the health practices described below.

Factors which have definitely been shown to increase life expectancy, demonstrated in decades-long research in animals and also in human population studies: Maintaining a lean body mass index (being at the lean end of your desirable weight range) while maintaining a high level of nutrition. Staying slim through nutrient deprivation doesn’t extend life. Staying slim by eating a high quality diet does. (See Weight Loss if you are overweight).

No other physical means (not even exercise) is proven to increase life expectancy. (Exercise is known to increase health span, meaning the number of years that a person stays healthy. We have no proof that it increases total lifespan, however).

Diet And Lifestyle Recommendations

  • Diet: eat a diet high in nutrient-rich foods.
  • Achieve and maintain a normal weight. Overweight is the second biggest cause of premature aging and death. (Smoking is first).
  • Exercise regularly. 30 minutes, 3 times per week minimum.
  • Don’t smoke! Smoking is the single biggest cause of premature aging and death.
  • Follow the Ten Golden Rules of Good Health.

Primary Support

Additional Support

 

Rejuvenex

®

Skin Rejuvenation and Protection Cream

rejuvenates and protects the skin in one easy formula. I have found it difficult if not impossible to find a good cream that contains all of the important ingredients for quality skin care until now.

is the first cosmetic preparation that I am aware of to combine the finest, proven ingredients into one easy formula. You can try to duplicate all of these skin essentials yourself, but you will be working hard and paying far more for the individual preparations. (Believe me, I know from experience. That’s how I’ve been making my own cream until now!)

This cream is rich, wonderful, contains “all the right stuff” as listed below, and what’s more, and a little goes a long way. You need only apply it morning and evening to get great results. Both men and women will benefit.

Shipping Information

Orders are processed 5 days a week, Monday through Friday, from 9 to 5 Arizona time. We try to get orders received by noon shipped out that same day – otherwise we’ll make every effort to get your order out the next business day if it is received after noon our time.

Orders are usually shipped within 48 business hours (2 days) via United States Postal Service Priority (USPS) Mail with Delivery Confirmation. You should receive your order within 3-4 business days.

If your order contains a “Concierge” product (special order) or back-order, your order will be shipped within 2 business days (48 hours) and the additional Concierge products or back-ordered items will be sent as soon as they arrive, at our expense. Your initial package will contain information which lists the “anticipated delay” of back-ordered or Concierge products.

 

Vitamin D A Special HealthBeat News Report


Vitamin D – You have been reading about it in the news, and you have wondered what is real and what is hype.

Dr. Myatt and Nurse Mark have researched and prepared this special report for HealthBeat News Readers.

Vitamin D — The Short Course

1.) Vit D is produced in our bodies in response to sun exposure. Vit D is also available from food and supplements.

2.) Vit D is FAR more important to health than was previously realized. I’m talking FAR more important.

3.) Vit D deficiency is widespread, including North America, even in sunny climates like Arizona. Many people who think they are getting enough Vitamin D from sunlight are mistaken.

4.) How to Optimize Vit D Levels for Good Health:

I.)  Vit D test, supplement accordingly, re-test

II.) Supplement at 5,000IU for 3 months, then test your levels.

III.) Don’t test, run the risk of being deficient, but take at least 2,000IU total per day. (This is still an extremely conservative dose, but much higher than the RDA of 400IU which hasn’t been changed yet to reflect the newer findings about Vit D). 

5.) Natural ways to obtain Vit D: Foods, supplements and sun exposure.

Vitamin D — Nutrient of the Decade: Are You Getting Enough?

The Consequences of Low Vitamin D

Vitamin D is called “the sunshine Vitamin” because our bodies make it in response to sun exposure.

Vit D is necessary for normal bone formation in both children and adults. In children, deficiencies of Vit D lead to rickets. In adults, deficiencies are associated with osteoporosis and osteomalacia (soft bones), decreased muscle strength and increased risk of fall. (1,12,14,22,43-48)Until recently, the bone-protecting effect was  about all that Vit D was known for, but the past decade of medical research has changed all that.

The newly appreciated Vitamin D deficiency risks include:

1.) heart disease: myocardial infarction, high blood pressure, heart failure, myopathy, sudden cardiac death, stroke (11,13-26, 30, 49-50)

2.) blood sugar problems: glucose intolerance, diabetes mellitus, metabolic syndrome (13-14,19,23-24,27-29)

3.) cancer prevention and improved cancer survival rates (7,8,11,14,15,24,31-37)

4.) upper respiratory tract infections, influenza and tuberculosis (24,30,38)

5.) cognitive impairment and low mood (38-40)

6.) autoimmune disease (multiple sclerosis, RA, systemic lupus erythromatosis (SLE) (15,24,26,29,30,32,41,42)

7.) misc. diseases: psoriasis, polycystic ovarian syndrome, inflammatory bowel disease

8.) urinary incontinence (54)

9.) and all-cause mortality! (5,6,7,24,30,51)

How “significant” are these associations? Here are some of the conclusions of various studies and meta-analyses (lots of studies looked at together) concerning Vit D. Italics are mine for emphasis.

“Research strongly supports the view … Vitamin D status would have significant protective effects against the development of cancer …. cancers of the breast, colon, prostate, ovary, lungs, and pancreas…” (8)

“High levels of Vitamin D among middle-age and elderly populations are associated with a substantial decrease in cardiovascular disease, type 2 diabetes and metabolic syndrome.” (9)

“Low levels of [Vitamin D] are independently predictive for fatal strokes” (10)

“It is estimated that there is a 30 to 50% reduction in risk for developing colorectal, breast, and prostate cancer by either increasing Vitamin D intake or increasing sun exposure…” (11)

“Oral Vitamin D supplementation between 700 to 800 IU/d appears to reduce the risk of hip and any nonvertebral fractures in ambulatory or institutionalized elderly persons” (12)

” 28 studies including 99,745 participants … highest levels of serum [Vit D] were associated with a 43% reduction in cardiometabolic disorders (cardiovascular disease, diabetes and metabolic syndrome) …” (9)

Are Your Vitamin D Levels Optimal? (Vitamin D Deficiency is Widespread)

One billion people worldwide are estimated to be Vit D deficient, and the problem affects us here in the United States as well. (2) One study found that more than half of North American women receiving drugs for prevention or treatment of osteoporosis were Vitamin D deficient. (1) Another study found 48% of pre-adolescent girls to be Vit D deficient (3). Other studies have found that 40% to 100% of older men and women in both the United States and Europe are Vitamin D deficient.[2] Because of the importance of Vit D and how widespread Vit D deficiency is, an estimated $100 to $200 billion is spent (wasted) each year on diseases which may really just be Vitamin D deficiencies. [4]

Age, overweight, dark skin color, use of sunscreen, and overprotection from the sun’s rays are causes of decreased production of Vit D in response to sunlight. (52,52)

How Much Vitamin D Should You Take?

Ideally, you should take whatever amount of Vitamin D puts you in the “optimal” range. Since the amount will be highly variable depending on age, sex, race, weight, daily sun exposure and diet, there is no “one size fits all” answer. Instead, blood testing of Vitamin D levels and increasing intake until optimal levels are reached is the surest way to obtain optimal concentrations of Vitamin D in the body.

Deficiency Insufficiency Sufficiency * Optimal Excess (Toxicity) <20ng/ml 20-32ng/ml 32-100ng/ml 40-80ng/ml > 150ng/ml

* – conventional medicine says that 30 ng/ml is “sufficient.” Chart references (59-62)

At the wellness Club we believe the most accurate and effective way to embark on a program of Vit D supplementation is to perform a Vit D test, supplement Vit D in accordance with the results, and then re-test in 3 months at which time your daily doses of Vit D can be fine-tuned for maintenance. March (right now!) is the best time to test initially because Vit. D stores tend to be lowest in this month.

The Vitamin D Council, a non-profit group dedicated to Vitamin D research and education recommends people take 5,000 IU per day for 2-3 months, then perform a Vitamin D test. They then suggest adjusting the dosage so that blood levels are between 50-80 ng/mL (or 125-200 nM/L) year-round. (55)

Alternately, some people opt to supplement without knowing their initial Vit D levels. A dose of 2000IU is quite conservative but certainly safe for almost anyone. In cases of significant Vit D deficiency conservative dosing such as this may take considerable time to rebuild healthy stores of this important Vitamin.

For those who wish to calculate their own Vit D requirements, 100 IU of Vitamin D could be expected to raise blood level of 25(OH)D by 1 ng/ml. (11)

Can too much Vitamin D can be toxic? Research shows that massive doses may eventually cause toxicity. One source found that in adults a sustained intake of 50,000 IU daily could produce toxicity within a few months (58) and 40,000 IU per day in infants has been shown to produce toxicity within 1 to 4 months. (56) That is ten times the recommended dose for each of those age groups! Vitamin D testing is good insurance that will allow you to safely fine-tune your dosage to your actual needs. Be careful though, since not all testing is the same and lab references and standards vary – be sure that you are comparing “apples to apples” and obtaining useable results when you are tested.

The 25-hydroxyVitamin D blood test (25(OH)D blood test) is a test that measures the amount of calcidiol circulating in the blood. This is the most accurate measure of the amount of Vitamin D in the body. The Wellness Club offers Vitamin D testing – performed by a lab that adheres to standardized references and values so that you know what you are getting when you receive your results. This can is performed at home with a “spot” (finger stick) blood test. Other tests that require a blood draw are also available.

How to Get to Your Optimal Vitamin D Levels

Start Vitamin D supplementation eight to twelve weeks before testing. Dr. John Cannell of the Vitamin D Council suggests a starting dose of 1,000 IU per 25 pounds of body weight. For example, a 150 pound person would take 6,000 IU Vitamin D per day. (150 divided by 25 = 6; 1,000IU x 6 = 6,000). Maintain this dose for 8-12 weeks, then test.

This dose may or may not put you in the optimal target range, but it certainly won’t put you in any “toxic” range. Remember, most adults can safely take up to 10,000IU per day and still be far away from Vitamin D toxicity which typically appears at 40,000-50,000IU taken for several months.

Although this dose should theoretically put you in an optimal range, numerous personal variations alter Vitamin D requirements. Some people will need a higher dose than this calculation affords. However, taking the calculated dose should at least put you “in the ballpark” for optimal dosing.

When you test results come back, you can use the number to help you know whether or not you need to increase your Vit D dose and by how much. It is estimated that each 1,000 IU increase in supplemental Vitamin D will generally produce a 10 ng/ml increase in the Vitamin D blood level (8). If your test result shows that you are 10ng/ml below your target, increase daily Vit D intake by 1,000IU per day for a total of 7,000IU per day from the above example. Continue this dose and re-test in another 3 months to verify that you are now in your optimal range.

Congratulations! You have found your optimal daily Vitamin D intake needed to maintain optimal Vitamin D blood levels.

How to Obtain Vitamin D Naturally

Exposure to sun is the most natural way to boost Vit D levels. Medical scientists have found that the skin produces approximately 10,000 IU of Vitamin D in response to as little as 30 minutes of unprotected summer sun exposure. (57)

Vitamin D can be obtained from food too. Since rickets in children is such a crippling but preventable condition, governments have long encouraged the “fortification” of dairy products and breads and cereals with token amounts of Vitamin D. In the United States and Canada, for example, fortified milk typically provides 100 IU per glass.

It is difficult to obtain optimal levels of Vitamin D from food alone.

Food IUs per serving* Percent DV** Cod liver oil, 1 tablespoon 1,360 340 Salmon (sockeye), cooked, 3 ounces 794 199 Mushrooms that have been exposed to ultraviolet light to increase vitamin D, 3 ounces (not yet commonly available) 400 100 Mackerel, cooked, 3 ounces 388 97 Tuna fish, canned in water, drained, 3 ounces 154 39 Milk, nonfat, reduced fat, and whole, vitamin D-fortified, 1 cup 115-124 29-31 Orange juice fortified with vitamin D, 1 cup (check product labels, as amount of added vitamin D varies) 100 25 Yogurt, fortified with 20% of the DV for vitamin D, 6 ounces (more heavily fortified yogurts provide more of the DV) 80 20 Margarine, fortified, 1 tablespoon 60 15 Sardines, canned in oil, drained, 2 sardines 46 12 Liver, beef, cooked, 3.5 ounces 46 12 Ready-to-eat cereal, fortified with 10% of the DV for vitamin D, 0.75-1 cup (more heavily fortified cereals might provide more of the DV) 40 10 Egg, 1 whole (vitamin D is found in yolk) 25 6 Cheese, Swiss, 1 ounce 6 2 *IUs = International Units.

**DV = Daily Value. DVs were developed by the U.S. Food and Drug Administration to help consumers compare the nutrient contents of products within the context of a total diet. The DV for vitamin D is 400 IU for adults and children age 4 and older. Food labels, however, are not required to list vitamin D content unless a food has been fortified with this nutrient.

Table courtesy of the U.S. Government National Institutes of Health Office of Dietary Supplements

Although cod liver oil is high in Vitamin D, it is also high in Vitamin A which interferes with Vit D uptake, so cod liver oil is not the best supplemental form of Vit D. Keep daily intake of pre-formed Vitamin A to a maximum of 5,000IU per day so as not to interfere with Vitamin D absorption. Beta carotene does not appear to interfere with Vit. D uptake.

Vegetarians need to be sure they are getting plenty of sunshine, because other than tiny amounts that may be found in UV-irradiated mushrooms, there are no vegetable sources of Vitamin D.

The Bottom Line on Vitamin D

Achieving Optimal Vitamin D  levels appears to be one of the most important things we can do for our overall health and life expectancy.

Please click on the image below enjoy an interesting and instructive video which discusses the relationship between Vitamin D and Cancer.

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References

1.) Holick MF, Siris ES, Binkley N, et al. Prevalence of Vitamin D inadequacy among postmenopausal North American women receiving osteoporosis therapy. J Clin Endocrinol Metab. 2005;90: 3215-3224.
2.) Holick MF. Vitamin D deficiency. N Engl J Med. 2007;357:266-281.
3.) Sullivan SS, Rosen CJ, Halteman WA, Chen TC, Holick MF. Adolescent girls in Maine at risk for Vitamin D insufficiency. J Am Diet Assoc. 2005;105:971-974.
4.) GrassrootsHealth. The Vitamin D deficiency epidemic. A call to D*action. http://www.grassrootshealth.org/daction/epidemic.php. Accessed May 8, 2009.
5.) GrassrootsHealth. Disease incidence prevention by serum 25(OH)D level. http://www.grassrootshealth.org/_download/disease_incidence_prev_chart_101608.pdf. Accessed May 8, 2009.
6.) Autier P, Gandini S. Vitamin D supplementation and total mortality. Arch Intern Med. 2007;167(16):1730-1737.
7.) Thomas L. Lenz. Vitamin D Supplementation and Cancer Prevention. Am J Lifestyle Med. 2009;3(5):365-368.
8.) Ingraham BA, Bragdon B, Nohe A. Molecular basis of the potential of Vitamin D to prevent cancer. Curr Med Res Opin. 2008 Jan;24(1):139-49.
9.) Parker J, Hashmi O, Dutton D, Mavrodaris A, Stranges S, Kandala NB, Clarke A, Franco OH. Levels of Vitamin D and cardiometabolic disorders: systematic review and meta-analysis. Maturitas. 2010 Mar;65(3):225-36. Epub 2009 Dec 23.
10.) Pilz S, Dobnig H, Fischer JE, Wellnitz B, Seelhorst U, Boehm BO, März W. Low Vitamin d levels predict stroke in patients referred to coronary angiography. Stroke. 2008 Sep;39(9):2611-3. Epub 2008 Jul 17.
11.) Holick MF. Vitamin D and sunlight: strategies for cancer prevention and other health benefits. Clin J Am Soc Nephrol. 2008 Sep;3(5):1548-54. Epub 2008 Jun 11.
12.) Bischoff-Ferrari HA, Willett WC, Wong JB, Giovannucci E, Dietrich T, Dawson-Hughes B. Fracture prevention with Vitamin D supplementation: a meta-analysis of randomized controlled trials. JAMA. 2005 May 11;293(18):2257-64.
13.) Anagnostis P, Athyros VG, Adamidou F, Florentin M, Karagiannis A. Vitamin D and Cardiovascular Disease: A Novel Agent for Reducing Cardiovascular Risk ? Curr Vasc Pharmacol. 2010 Feb 25. [Epub ahead of print]
14.) Holick MF. Vitamin D: importance in the prevention of cancers, type 1 diabetes, heart disease, and osteoporosis. Am J Clin Nutr. 2004 Mar;79(3):362-71.
15.) Holick MF. Vitamin D and sunlight: strategies for cancer prevention and other health benefits. Clin J Am Soc Nephrol. 2008 Sep;3(5):1548-54. Epub 2008 Jun 11.
16.) Judd SE, Tangpricha V. Vitamin D deficiency and risk for cardiovascular disease. Am J Med Sci. 2009 Jul;338(1):40-4.
17.) Kendrick J, Targher G, Smits G, Chonchol M.25-HydroxyVitamin D deficiency is independently associated with cardiovascular disease in the Third National Health and Nutrition Examination Survey. Atherosclerosis. 2009 Jul;205(1):255-60. Epub 2008 Nov 11.
18.) Lee W, Kang PM. Vitamin D deficiency and cardiovascular disease: Is there a role for Vitamin D therapy in heart failure? Curr Opin Investig Drugs. 2010 Mar;11(3):309-14.
19.) Martins D, Wolf M, Pan D, Zadshir A, Tareen N, Thadhani R, Felsenfeld A, Levine B, Mehrotra R, Norris K. Prevalence of cardiovascular risk factors and the serum levels of 25-hydroxyVitamin D in the United States: data from the Third National Health and Nutrition Examination Survey. Arch Intern Med. 2007 Jun 11;167(11):1159-65.
20.) McConnell JP, Foley KF, Vargas GM. HypoVitaminosis D: a new risk marker for cardiovascular disease. Clin Lab Sci. 2009 Fall;22(4):240-6.
21.) Mertens PR, Müller R. Vitamin D and cardiovascular risk. Int Urol Nephrol. 2009 Dec 29. [Epub ahead of print]
22.) Murlikiewicz K, Zawiasa A, Nowicki M. Vitamin D–a panacea in nephrology and beyond] Pol Merkur Lekarski. 2009 Nov;27(161):437-41.{article in Polish]
23.) Parker J, Hashmi O, Dutton D, Mavrodaris A, Stranges S, Kandala NB, Clarke A, Franco OH. Levels of Vitamin D and cardiometabolic disorders: systematic review and meta-analysis. Maturitas. 2010 Mar;65(3):225-36. Epub 2009 Dec 23.
24.) Pilz S, Dobnig H, Nijpels G, Heine RJ, Stehouwer CD, Snijder MB, van Dam RM, Dekker JM. Vitamin D and mortality in older men and women. Clin Endocrinol (Oxf). 2009 Nov;71(5):666-72. Epub 2009 Feb 18.
25.) Pilz S, März W, Wellnitz B, Seelhorst U, Fahrleitner-Pammer A, Dimai HP, Boehm BO, Dobnig H. Association of Vitamin D deficiency with heart failure and sudden cardiac death in a large cross-sectional study of patients referred for coronary angiography. J Clin Endocrinol Metab. 2008 Oct;93(10):3927-35. Epub 2008 Aug 5.
26.) Wu PW, Rhew EY, Dyer AR, Dunlop DD, Langman CB, Price H, Sutton-Tyrrell K, McPherson DD, Edmundowicz D, Kondos GT, Ramsey-Goldman R. 25-hydroxyVitamin D and cardiovascular risk factors in women with systemic lupus erythematosus. Arthritis Rheum. 2009 Oct 15;61(10):1387-95.
27.) Baz-Hecht M, Goldfine AB. The impact of Vitamin D deficiency on diabetes and cardiovascular risk. Curr Opin Endocrinol Diabetes Obes. 2010 Apr;17(2):113-9.
28.) Cheng S, Massaro JM, Fox CS, Larson MG, Keyes MJ, McCabe EL, Robins SJ, O’Donnell CJ, Hoffmann U, Jacques PF, Booth SL, Vasan RS, Wolf M, Wang TJ. Adiposity, cardiometabolic risk, and Vitamin D status: the Framingham Heart Study. Diabetes. 2010 Jan;59(1):242-8. Epub 2009 Oct 15.
29.) Holick MF. Sunlight and Vitamin D for bone health and prevention of autoimmune diseases, cancers, and cardiovascular disease. Am J Clin Nutr. 2004 Dec;80(6 Suppl):1678S-88S.
30.) Ginde AA, Scragg R, Schwartz RS, Camargo CA Jr. Prospective study of serum 25-hydroxyVitamin D level, cardiovascular disease mortality, and all-cause mortality in older U.S. adults. J Am Geriatr Soc. 2009 Sep;57(9):1595-603. Epub 2009 Jun 22.
31.) Grant WB. How strong is the evidence that solar ultraviolet B and Vitamin D reduce the risk of cancer?: An examination using Hill’s criteria for causality. Dermatoendocrinol. 2009 Jan;1(1):17-24.
32.) Holick MF, Chen TC. Vitamin D deficiency: a worldwide problem with health consequences. Am J Clin Nutr. 2008 Apr;87(4):1080S-6S.
33.) Holick MF. Vitamin D: its role in cancer prevention and treatment. Prog Biophys Mol Biol. 2006 Sep;92(1):49-59. Epub 2006 Mar 10.
34.) Ingraham BA, Bragdon B, Nohe A. Molecular basis of the potential of Vitamin D to prevent cancer. Curr Med Res Opin. 2008 Jan;24(1):139-49.
35.) Pilz S, Dobnig H, Winklhofer-Roob B, Riedmüller G, Fischer JE, Seelhorst U, Wellnitz B, Boehm BO, März W. Low serum levels of 25-hydroxyVitamin D predict fatal cancer in patients referred to coronary angiography. Cancer Epidemiol Biomarkers Prev. 2008 May;17(5):1228-33. Epub 2008 May 7.
36.) Pilz S, Tomaschitz A, Obermayer-Pietsch B, Dobnig H, Pieber TR. Epidemiology of Vitamin D insufficiency and cancer mortality. Anticancer Res. 2009 Sep;29(9):3699-704.
37.) Ginde AA, Mansbach JM, Camargo CA Jr. Association between serum 25-hydroxyVitamin D level and upper respiratory tract infection in the Third National Health and Nutrition Examination Survey. Arch Intern Med. 2009 Feb 23;169(4):384-90.
38.) Annweiler C, Schott AM, Allali G, Bridenbaugh SA, Kressig RW, Allain P, Herrmann FR, Beauchet O. Association of Vitamin D deficiency with cognitive impairment in older women: cross-sectional study. Neurology. 2010 Jan 5;74(1):27-32. Epub 2009 Sep 30.
39.) Cherniack EP, Troen BR, Florez HJ, Roos BA, Levis S. Some new food for thought: the role of Vitamin D in the mental health of older adults. Curr Psychiatry Rep. 2009 Feb;11(1):12-9.
40.) Wilkins CH, Sheline YI, Roe CM, Birge SJ, Morris JC. Vitamin D deficiency is associated with low mood and worse cognitive performance in older adults. Am J Geriatr Psychiatry. 2006 Dec;14(12):1032-40.
41.) Cutolo M, Otsa K. Review: Vitamin D, immunity and lupus. Lupus. 2008;17(1):6-10.
42.) Kamen DL, Cooper GS, Bouali H, Shaftman SR, Hollis BW, Gilkeson GS. Vitamin D deficiency in systemic lupus erythematosus. Autoimmun Rev. 2006 Feb;5(2):114-7. Epub 2005 Jun 21.
43.) Bischoff-Ferrari HA, Willett WC, Wong JB, Giovannucci E, Dietrich T, Dawson-Hughes B. Fracture prevention with Vitamin D supplementation: a meta-analysis of randomized controlled trials. JAMA. 2005 May 11;293(18):2257-64.
44.) Bischoff HA, Stähelin HB, Tyndall A, Theiler R. Relationship between muscle strength and Vitamin D metabolites: are there therapeutic possibilities in the elderly? Z Rheumatol. 2000;59 Suppl 1:39-41.
45.) DIPART (Vitamin D Individual Patient Analysis of Randomized Trials) Group. Patient level pooled analysis of 68 500 patients from seven major Vitamin D fracture trials in US and Europe. BMJ. 2010 Jan 12;340:b5463. doi: 10.1136/bmj.b5463.
46.) Houston DK, Cesari M, Ferrucci L, Cherubini A, Maggio D, Bartali B, Johnson MA, Schwartz GG, Kritchevsky SB. Association between Vitamin D status and physical performance: the InCHIANTI study. J Gerontol A Biol Sci Med Sci. 2007 Apr;62(4):440-6.
47.) Kwon J, Suzuki T, Yoshida H, Kim H, Yoshida Y, Iwasa H. Concomitant lower serum albumin and Vitamin D levels are associated with decreased objective physical performance among Japanese community-dwelling elderly. Gerontology. 2007;53(5):322-8. Epub 2007 May 29.
48.) Pfeifer M, Begerow B, Minne HW. Vitamin D and muscle function. Osteoporos Int. 2002 Mar;13(3):187-94.
49.) Judd SE, Nanes MS, Ziegler TR, Wilson PW, Tangpricha V. Optimal Vitamin D status attenuates the age-associated increase in systolic blood pressure in white Americans: results from the third National Health and Nutrition Examination Survey. Am J Clin Nutr. 2008 Jan;87(1):136-41.
50.) Pilz S, Dobnig H, Fischer JE, Wellnitz B, Seelhorst U, Boehm BO, März W. Low Vitamin d levels predict stroke in patients referred to coronary angiography. Stroke. 2008 Sep;39(9):2611-3. Epub 2008 Jul 17.
51.) Melamed ML, Michos ED, Post W, Astor B.25-hydroxyVitamin D levels and the risk of mortality in the general population. Arch Intern Med. 2008 Aug 11;168(15):1629-37.
52.) Jacobs ET, Alberts DS, Foote JA, Green SB, Hollis BW, Yu Z, Martínez ME. Vitamin D insufficiency in southern Arizona. Am J Clin Nutr. 2008 Mar;87(3):608-13.
53.) Park S, Johnson MA. Living in low-latitude regions in the United States does not prevent poor Vitamin D status. Nutr Rev. 2005 Jun;63(6 Pt 1):203-9.
54.) Low Vitamin D Levels Tied to Incontinence. WebMD March 22, 2010 http://www.webmd.com/urinary-incontinence-oab/news/20100322/low-Vitamin-d-linked-incontinence.
55.) The Vitamin D Council. Vitamin D Council
56.) Wikipedia: Vitamin D. Wikipedia Vitamine D
57.) Holick MF. Environmental factors thatinfluence the cutaneous production of Vitamin D. Am J Clin Nutr. 1995 Mar;61(3 Suppl):638S-645S.
58.) Vieth R. Vitamin D supplementation, 25-hydroxyVitamin D concentrations, and safety. Am J Clin Nutr. 1999 May;69(5):842-56.
59.) Holick MF. Vitamin D deficiency. N Engl J Med. 2007;357:266-281.
60.) GrassrootsHealth. Disease incidence prevention by serum 25(OH)D level.Grassroots Heaalth. Accessed May 8, 2009.
61.) Dall T, Anderson J. Vitamin D: merging research into clinical lipid practice. Lipid Spin. 2008;6(3):4-8.
62.) Heaney RP. What is a Vitamin D deficiency?Grassroots Health Vitamin D deficiency. Accessed May 8, 2009.

 

 

Xylitol

The Sweet That’s Good For You

Xylitol Chewing GumUnique Sweet® Xylitol Gum, Peppermint

Unique Sweet® Gum with xylitol, is a low-calorie delicious-tasting gum that freshens your breath, and can fight cavities and plaque. Xylitol is a natural sweetener found in strawberries, raspberries and plums. Unlike sugar, xylitol is actually good for teeth. It’s beneficial for diabetics, it stimulates remineralization of teeth, and it fights ear and sinus infections.

Recommended Dosage: chew Unique Sweet® gum after each meal or snack.

Currently Unavailable – Sorry!

Nutrition Facts – Unique Sweet Gum – Serving Size: 1 piece (1.08 g)   Amount Per Serving % Daily Value Calories: 1.7   Total Fat 0 g 0% Total Carbohydrates .72 g <1% Sugars 0 g **   Xylitol .72 g **   Sodium 0 g 0% Protein 0 g 0% *Percent Daily Values (DV) are based on a 2000 calorie diet. Ingredients: Xylitol, gum base, natural flavors, glycerin, gum arabic, soy lecithin and beeswax. Contains: Soy

Xylitol Nasal WashXlear Xylitol Nasal Wash

Aids in relief of nasal irritation caused by pollutants, allergens, and infections of the nasal passages. Safe for infants. Non-habit forming.

Recommended Dosage: For best results use at least two times per day.

Ingredients: Purified Water, Xylitol, Saline, and grapefruit seed as a preservative.

Click here to
Order This Concierge Product WellevateThis is a separate website and you will need to create a new account to order.

Oral ChelatoRx

Safe and Effective Alternative to IV Chelation Therapy

Oral ChelatoRx is an EDTA-containing formula designed to provide oral chelation. This formula chelates or “binds” heavy metals and excessive levels of calcium and other metal compounds from the bloodstream. Many anti-atherosclerotic protocols and toxic metal reducing protocols benefit from the inclusion of this product. Be sure to take an optimal mineral-containing formula such as Maxi Multi to insure replacement of the “good” minerals during oral chelation therapy.

Here are some of the known actions of Oral ChelatoRx’ Ingredients:

EDTA (ethylene diamine tetraacetic acid) is a synthetic amino acid, approved by the FDA as a pharmaceutical agent for the treatment of lead and other heavy metal poisoning or exposure. Prior to the late 1970’s, the FDA also approved EDTA as being “possibly effective in occlusive vascular disorders…arrhythmias and atrioventricular induction defects…and in the treatment of pathologic conditions to which calcium tissue deposits or hypercalcemia may contribute other than those listed above.”  Although they have since withdrawn the latter “indication” (for reasons that were never substantiated by any science that I can find), many physicians still use EDTA chelation to bind and remove toxic metals and abnormal calcium deposits in the body.

Although best known in “IV chelation therapy” for atherosclerosis, low-dose daily oral EDTA also appears to be safe and effective. Clinical studies with EDTA reported loss of fat in rats, reduction of cholesterol in rabbits, and reduced blood pressure in humans. Human studies of oral EDTA have shown significant improvements in blood pressure, cholesterol levels, intermittent claudication and angina. In another study, chest pain symptoms completely resolved in patients taking oral EDTA for 3 months.

EDTA is considered quite safe and is approved as a food additive in the US.

Chlorella is a potent detoxifier, high in minerals and phytonutrients. Have you ever seen green algae growing in stagnant water? Algae is nature’s method of cleaning up impure water. Chlorella, an algae, does the same thing in the human body: it pulls out toxins and impurities. Chlorella is known to eliminate toxins, pesticides, and heavy metals from the body.

Garlic: Many studies have shown that garlic helps the cardiovascular system. Some trials show that garlic lowers cholesterol and triglyceride levels. Garlic also inhibits platelet aggregation and increases fibrinolysis, which decreases “blood stickiness.” Garlic has blood-pressure lowering and has antioxidant activity.

Garlic’s heart-protective effects were seen in a four-year clinical trial on people 50–80 years old with atherosclerosis, where consumption of 900 mg of standardized garlic supplement reduced arterial plaque formation by 5–18%.

Malic Acid is a naturally occurring compound that plays an important role in deriving adenosine triphosphate (ATP; the “energy currency” of the body) from food. It is found in a wide variety of fruits and vegetables, but the richest source is apples, which is why malic acid is sometimes referred to as “apple acid.” ATP is crucial to normal heart function.

Gugulipid (Commiphora mukul) is a small, thorny plant found in India. The resin contains compounds that lower cholesterol and triglyceride levels, including total cholesterol, LDL and VLDL cholesterols ( the “bad” cholesterols). At the same time, gugul raises HDL cholesterol (the “good” cholesterol). Gugul contains antioxidants which prevents LDL cholesterol from oxidizing, an action that helps protect against atherosclerosis. Guggul also reduces the stickiness of platelets—another effect that lowers the risk of coronary artery disease.

Serrapeptase is an enzyme derived from the Serratia bacteria which lives in the intestinal tract of silkworms. It has been used for over 30 years in Europe and Asia (where it is approved as a “drug”) to reduce pain and inflammation. Serrapeptase dissolves blood clots and prevents abnormal blood clotting.

Suggested dose: 12 capsules per day in a single dose between meals or as recommended by a physician.

Supplement Facts Serving Size: 6 Capsules Servings Per Container: 60 alt Amount Per Serving % Daily Value alt Calcium 70mg   7% (as calcium disodium EDTA) alt Magnesium 206 mg 51% (as 85% magnesium aspartate, 15% magnesium orotate) alt Sodium 80 mg 3% as calcium disodium EDTA alt Potassium 125 mg 4% (as 75% potassium aspartate and 25% potassium orotate) alt EDTA 500 mg * (as calcium disodium ethylenediaminetetraacetic acid) alt Chlorella 375 mg * (Chlorella regularis) alt Garlic 313 mg * (Allium sativum) extract (root) 10,000 ppm allicin alt Malic Acid 300 mg * alt Gugulipid 250 mg * (Commiphora mukul) extract (gum) 2.5% guggulsterones alt Serrapeptase enzyme (30,000 IU activity) 15 mg * alt *Daily Value not established Other Ingredients:
Hydroxypropyl methylcellulose (Vcap) and magnesium stearate.

Contains no added starch, salt, wheat, gluten,
corn, coloring, or dairy products.

Keep container tightly closed in a cool,
dry and dark place. Keep out of reach of children.

References

1.)Calcium disodium edetate and disodium edetate. Federal Register, Volume 35, No. 8, Tuesday, January 13, 1970, 585-587.
2.) Perry, H. Mitchell, Schroeder, Henry A. Depression of cholesterol levels in human plasma following ethylenediamine tetracetate and hydralazine. J Chronic Diseases, 1955, 2: 5, 520-532.
3.) Foreman, H., Trujillo, T. The metabolism of C14 labeled ethylenediaminetetraacetic acid in human beings. J Lab Clin Med, 1954, 43: 566-571.
4.) Born, G.R., and Geurkink, T.L. Improved peripheral vascular function with low dose intravenous ethylene diamine tetraacetic acid (EDTA). Townsend Letter for Doctors. July, 1994, # 132, 722-726.
5.) Mariani, B., Bisetti, A., and Romeo, V. Blood-cholesterol-lowering action of the sodium salt of calciumethylenediaminotetraacetic acid. Gazz Intern Med e Chir, 1957. 62: 1812-1823.
6.) Gordon, G. Oral Chelation with EDTA. J Holistic Medicine, 1986, 8: 1 & 2, 79-80.
7.) Warshafsky S, Kamer R, Sivak S. Effect of garlic on total serum cholesterol: A meta-analysis. Ann Int Med 993;119:599–605.
8.) Silagy C, Neil A. Garlic as a lipid-lowering agent—a meta-analysis. J R Coll Phys London 1994;28:39–45.
9.) Neil HA, Silagy CA, Lancaster T, et al. Garlic powder in the treatment of moderate hyperlipidaemia: A controlled trial and a meta-analysis. J R Coll Phys 1996;30:329–34.
10.) Legnani C, Frascaro M, Guazzaloca G, et al. Effects of a dried garlic preparation on fibrinolysis and platelet aggregation in healthy subjects. Arzneim-Forsch Drug Res 1993;43:119–22.
11.) Silagy CA, Neil HA. A meta-analysis of the effect of garlic on blood pressure. J Hyperten 1994;12:463–8.
12.) Kleijnen J, Knipschild P, Ter Riet G. Garlic, onion and cardiovascular risk factors: A review of the evidence from human experiments with emphasis on commercially available preparations. Br J Clin Pharmacol 1989;28:535–44.
13.) Koscielny J, Klüendorf D, Latza R, et al. The antiatherosclerotic effect of Allium sativum. Atherosclerosis 1999;144:237–49.
14.) Satyavati GV. Gum guggul (Commiphora mukul)—The success of an ancient insight leading to a modern discovery. Indian J Med 1988;87:327–35.
15.) Nityanand S, Kapoor NK. Hypocholesterolemic effect of Commiphora mukul resin (Guggal). Indian J Exp Biol 1971;9:367–77.
16.) Singh K, Chander R, Kapoor NK. Guggulsterone, a potent hypolipidaemic, prevents oxidation of low density lipoprotein. Phytother Res 1997;11:291–4.
17.) Mester L, Mester M, Nityanand S. Inhibition of platelet aggregation by guggulu steroids. Planta Med 1979;37:367–9.
18.) Heinrich, J. et al. “Fibrinogen and factor VII in the prediction of coronary risk.” Arterioscler Thromb 1994, 14:54-59.
19.) Hager, K. et al. “Fibrinogen and Aging.” Aging (Milano) 1994, 6:133-38.
20.) Montalescot, G. et al. “Fibrinogen as a risk factor for coronary heart disease.” Eur Heart J 1998, 19 Suppl H:H11-17.

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Dr. Myatt’s Pain Drawing

Dr. Myatt’s Pain Drawing

Please draw in the appropriate location of pain with the symbol that best describes the discomfort that you are presently experiencing. Be as accurate and complete as you can. Then answer the questions below. Patient Name:

Date:

Sharp and Stabbing = ++++++
Dull and Achy = VVVVV
Pins and Needles = OOOOO
Numbness = ////////////
Spasm or Cramp = SSSSS
Stiff or Tight or Tense = XXXXX

 

 

 

 

 

 

What makes the pain better?

What makes it worse?

Have you ever had this pain before?

If yes, when?

When did the pain begin?

Why did the pain begin? (If you know)

How often do you have the pain?
A – Occasionally
B – Half the time
C – Frequently
D – Constantly

How intense is the pain?
1 – slight
2 – Mild
3 – Moderate
4 – Severe

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PARASITES


Natural Solutions To Deal With Parasitic Infections

Symptoms of acute parasite infection are usually obvious, but “sub-acute” (low grade infection) of parasites can cause or contribute to many health problems and the diagnosis is often missed in conventional medicine (for reasons which I describe below). Symptoms of sub-acute parasitic infection can include:

  • chronic GI symptoms (IBS, diarrhea, intestinal cramps, constipation)
  • chronic digestive complaints (belching, heartburn, malabsorption)
  • headaches
  • weight loss (unintended)
  • autoimmune disease
  • multiple food intolerances
  • chronic fatigue
  • fever, chills (especially if no other cause is found)

Parasites are, in the broadest sense, any organism that lives on or in another organism and detracts from the health and vigor of the host. An organism that lives on or in another organism but does NOT detract from the health and vigor of the host is referred to as a commensal. If the organisms benefit from their relationship with each other, they are known as symbiots and their relationship is called symbiosis.

Obviously, many relationships between organisms and humans can be considered parasitic: virus/human, bacteria/human, worm/human, even human/human! Medically speaking, the term parasite is most commonly understood to mean the relationship between a human host and a protozoa or worm. These relationships are almost always destructive to the host. In addition, certain bacteria, fungi, protozoa and amoebas have dual relationships with the host and can be either parasitic or commensal.

Because of their parasitic potential, parasite/commensals will be considered in addition to true parasite. Some examples of each include:

True Parasites

Protozoan: E. histolytica, Giardia lamblia, Plasmodium (4 species), Leshmania, Toxoplasmosis, Cryptosporidia

Worms: Enterobiasis (pinworm), Trichuriasis (whipworm), Ascariasis, Necator americanus (hookworm), Strongyloidiasis (threadworm), Trichinella, Wucheria bancroftii

Parasite/Commensals

Protozoan: Blastocystis hominis, Dientamoeba fragilis

Amoeba: Endolimax nana, E. histolytica, E. coli, Iodamoeba butschlie

Fungi: Candida albicans, candida spp.

Bacteria: Klebsiella pneumonia

Symptoms of Acute Parasitic Infection

History and symptoms have largely been regarded as the guiding factors for diagnosis of parasites. These symptoms vary according to the species of organism, what part of the body is infected, and the severity of the infestation.

Systemic symptoms of fever, chills, skin lesions, hemolytic anemia or jaundice, especially following out-of-country travel, often suggest the diagnosis. Overt GI symptoms including diarrhea, abdominal pain, cramping, flatulence, epigastric pain, intermittent nausea and malodorous stools may indicate intestinal infection.

Holistic Consideration of Parasites

When history and/or symptoms are overt, a diagnosis of parasites may be readily suspected. Sub-acute infections resulting in low-grade GI symptoms are encountered routinely in general family practice but are frequently unrecognized as such. This problem of under-diagnosis is likely due to several factors:

  1. Lack of history of exposure. It is not necessary to travel out of the country to acquire a parasite. Many organisms present themselves in food. A history of world travel used to be a major factor guiding physicians to diagnosis. Today, however, it must be appreciated that any non-exotic parasite can be acquired locally.
  2. Sub-acute nature of symptoms: Acute parasitic infections, with attendant severity of GI or systemic symptoms, is often easier to diagnose than sub-acute infection. Sub-acute infection can be either caused by a true parasite or by a parasite/commensal and can trigger a variety of local complaints that are not typically thought of in conjunction with parasite, but should be.
  3. Inadequacy of laboratory evaluation. The first “weak link” in the diagnostic chain, especially in sub-acute infections, is often the physician. The second weak link can be the medical lab, the method of collection, or both.

Many medical labs are equipped to identify overt parasitism, especially when the specimen yield is high. When the percent yield is low, the organism is often missed. Further, exotic species are more often recognized than non-exotic species and parasitic/commensal organisms, which may be overlooked or under-reported. For example: Candida albicans, is rarely reported on a conventional stool assay because it is considered a commensal and therefore not thought of as infectious. However, an overgrowth of this organism is known to behave in parasitic fashion to the host.

What To Do If You Suspect Parasites

First, don’t “play doctor” unless you ARE one! See a physician about your complaint and get a “work-up” by conventional medical standards. This will probably include blood and perhaps urine analysis and physical diagnosis. IF nothing can be identified to explain your complaint AND you have symptoms on the list above, then it is time to have some additional testing performed by an alternative medicine physician to evaluate for a sub-acute parasitic infection. You need a physician consult for this.

Based on your symptoms, the appropriate tests FROM THE RIGHT LAB will be ordered. (Remember, many labs miss the diagnosis when the number of parasites present in the sample are small). I use laboratories that specialize in looking for low-grade parasitic infections, so if something is there, they will find it. Also, increasing the number of specimens has been proven to dramatically increase the likelihood of finding an offending organism. This is because parasites in the GI tract “shed” only periodically and are often missed by a single stool sample.

The most common tests needed to discover parasites include the Comprehensive Digestive Stool Analysis, Candida testing and a Gastric Acid Self-Test. Again, knowing which tests to order is a matter of clinical judgment that few laymen are prepared to make.

If you believe, based on your chronic symptoms and from what you have just learned, that you may have parasites as a cause of your problems, please give me a call for consultation. I can help you learn if parasites are a cause of your “undiagnosed” complaint.

Exact treatments will differ depending on the organism and location. There are some basic recommendations that apply to all parasitic infections, however.

Primary Support

  • Maxi Multi: 3 caps, 3 times per day with meals. Parasites “sap” nutrients from the body and weaken the immune system. Especially important nutrients include vitamin A and B12, but any nutrient can be deficient based on the nutritional patterns of the parasite.
  • Fiber Formula: (helps transport killed organisms out of the body): 6 caps, 2 times per day between meals.
  • Chlorophyll: (water soluable; intestinal detoxifier): 1 cap, 2 times per day with meals.
  • Immune Support: 1 cap, 2 times per day with meals
  • SupremaDophilus: 1 cap before bed. Helps replace “friendly” gut bacteria.
  • Parasite Tincture: as directed by physician.
  • Berberine has been shown to have anti-protazoan effects

Additional Support

  • Colloidal Silver: 1-1/2 tsp. two times per day (for 140 pound body weight; adjust up or down as needed). Use 5ppm silver for 14 days.

    NOTE: Sub-acute parasitic infections are usually better treated by alternative medicine than by conventional drugs. The reason is that anti-parasitic medicines are toxic to the host as well as the parasite, and low-grade infections must be treated for a much longer duration of time to be sure that all organisms are killed.

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Help Yourself to Health!
with
Dana Myatt, N.M.D.
Family Practice, Natural Medicine

© 2005 Dr. Myatt’s Wellness Club