Search Results for: Well-Prepared COF-C02 Reliable Exam Topics - Professional COF-C02 Latest Dumps Questions - Excellent COF-C02 Valid Exam Topics 🐚 Search for ⮆ COF-C02 ⮄ on ➽ www.pdfvce.com 🢪 immediately to obtain a free download 🆚New COF-C02 Exam Practice

Fertility Restore Alpha Lipoic Acid 60 caps

Alpha Lipoic Acid

Neurological Antioxidant and Energy Transporter

Alpha Lipoic Acid chelates free iron from the forebrain, thereby protecting against free radical-induced brain aging. It improves mitochondrial function (the “energy producing units” of the cell) and works well in combination with CoQ10 and Acetyl-l-Carnitine to enhance energy production. Lipoic acid is also involved in the conversion of carbohydrates to energy. Use for:

  • overweight and obesity
  • brain health
  • neurological disease
  • cataract prevention
  • diabetes
  • heart disease
  • detoxification support

Each (one) capsule contains: Alpha Lipoic Acid – 100 mg

Suggested dose: 50-150mg per day (one capsule).

Product # 170 – Alpha Lipoic Acid – (60 capsules) $19.95

 

Berberine 500mg 60 capsules

Powerful effects on blood sugar, cholesterol, heart disease and cancer

Berberine is an alkaloid derived from various plants including Goldenseal (Hydrastis canadensis), Oregon grape (Berberis aquifolium), Barberry (Berberis vulgaris), Chinese Goldenthread (Coptis) and other plants. It has a bright yellow color.

Same potency, now in ONE capsule instead of two!
Previous formula: 120 caps per bottle, 2 caps = 500mg (60 doses per bottle)
Now: 60 caps per bottle, 1 cap = 500mg (60 doses per bottle)

Actions of Berberine:

Broad-spectrum anti-microbial: (1-6, 60)
Berberine has been found to be an effective against:

  • Chlamydia
  • Candida (multiple species)
  • E. coli
  • Salmonella typhi
  • Entamoeba histolytica
  • Staphylococcus aureus
  • Streptococcus sanguis
  • Pseudomonas aeruginosa
  • Helicobacter pylori
  • other Gram-positive bacteria and fungi
  • Some studies suggest that goldenseal may also be effective against viruses, fungi, protozoans, helminths (worms) and MRSA (methicillin-resistant Staphylococcus
    aureus)

Blood sugar regulation / diabetes type II. Some studies have shown that berberine lowers blood sugar as effectively as the diabetes drug metformin. (7-18)

Cholesterol-lowering effects. Studies show lipid-lowering effects on total cholesterol, triglyceride and LDL cholesterol. (19-26)

Cardic effects. Berbeine has been found to have anti-arrhythmic effects and may also be useful for CHF (congestive heart failure). (27-34)

Anti-cancer effects. Berberine has been found to have anti-neoplastic effects against a wide variety of cancer cell types including brain, bladder, breast, bone, colon, liver, lung, prostate, skin cancer and leukemia. (35-59)

Learn More about Berberine in our HealthBeat News article:
The “Swiss Army Knife” Of Supplements

Berberine 500 mg of Berberine per each capsules.

Suggested Use As a dietary supplement:

For antimicrobial effect: Take one capsule two times daily for up to fourteen (14) days with water at mealtimes. Not to be taken by pregnant or lactating women or children under 12 years old.

For blood sugar regulation: one capsule, 3 times daily with meals.

N361 – Berberine 500mg – 60 vegetarian caps – $34.97

Product description: Berberine 500mg 60 vegetarian caps

Ingredients per capsule:
Berberine HCL 500 mg (Extracted from Berberis Aristata, root)
Calcium (as calcium carbonate) 35mg

Other ingredients: Vegetarian capsule (hydroxypropyl methylcellulose and water) and silicone dioxide.

Capsule: Vegetable source from cellulose, preservative free.

Hypoallergenic: Contains NO yeast, wheat, gluten, soy protein, milk/dairy, corn, sodium, sugar, starch, artificial coloring, preservatives, or flavoring.

100% Vegetarian Formula

Bottle contains both a desiccant and an anti-oxidant packet to protect the contents from degradation due to moisture and oxidation

References

1.) Hahn FE, Ciak J. Berberine. Antibiotics 1976;3:57788.
2.) Kim SH, Lee SJ, Lee JH, Sun WS, Kim JH. Antimicrobial activity of 9-O-acyl- and 9-O-alkylberberrubine derivatives. Planta Med. 2002 Mar;68(3):277-81.
3.) Scazzocchio F, Cometa MF, Tomassini L, Palmery M. Antibacterial activity of Hydrastis canadensis extract and its major isolated alkaloids. Planta Med. 2001 Aug;67(6):561-4.
4.) Mahady GB, Pendland SL, Stoia A, Chadwick LR. In vitro susceptibility of Helicobacter pylori to isoquinoline alkaloids from Sanguinaria canadensis and Hydrastis canadensis.Phytother Res. 2003 Mar;17(3):217-21.
5.) [no authors listed]. Berberine. Altern Med Rev. 2000 Apr;5(2):175-7.
Berberine. Altern Med Rev. Apr 2000;5(2):175-177.
6.) Yu HH, Kim KJ, Cha JD, et al. Antimicrobial activity of berberine alone and in
combination with ampicillin or oxacillin against methicillin-resistant Staphylococcus
aureus. J Med Food. Winter 2005;8(4):454-461.
7.) Chen C, Zhang Y, Huang C. Berberine inhibits PTP1B activity and mimics
insulin action. Biochem Biophys Res Commun. Jul 2 2010;397(3):543-547.
8.) Turner N, Li JY, Gosby A, et al. Berberine and its more biologically available
derivative, dihydroberberine, inhibit mitochondrial respiratory complex I: a
mechanism for the action of berberine to activate AMP-activated protein kinase
and improve insulin action. Diabetes. May 2008;57(5):1414-1418.
9.) Lee YS, Kim WS, Kim KH, et al. Berberine, a natural plant product, activates
AMP-activated protein kinase with beneficial metabolic effects in diabetic and
insulin-resistant states. Diabetes. Aug 2006;55(8):2256-2264.
10.) Ma X, Egawa T, Kimura H, et al. Berberine-induced activation of 5’-adenosine
monophosphate-activated protein kinase and glucose transport in rat skeletal
muscles. Metabolism. Apr 26 2010.
11.) Hwang JT, Kwon DY, Yoon SH. AMP-activated protein kinase: a potential target
for the diseases prevention by natural occurring polyphenols. N Biotechnol. Oct
1 2009;26(1-2):17-22.
12.) Zhang H, Wei J, Xue R, et al. Berberine lowers blood glucose in type 2 diabetes
mellitus patients through increasing insulin receptor expression. Metabolism. Feb
2009;59(2):285-292.
13.) Kong WJ, Zhang H, Song DQ, et al. Berberine reduces insulin resistance
through protein kinase C-dependent up-regulation of insulin receptor expression.
Metabolism. Jan 2009;58(1):109-119.
14.) Yin J, Gao Z, Liu D, Liu Z, Ye J. Berberine improves glucose metabolism
through induction of glycolysis. Am J Physiol Endocrinol Metab. Jan
2008;294(1):E148-156.
15.) Lu SS, Yu YL, Zhu HJ, et al. Berberine promotes glucagon-like peptide-1
(7-36) amide secretion in streptozotocin-induced diabetic rats. J Endocrinol. Feb
2009;200(2):159-165.
16.) Yu Y, Liu L, Wang X, Liu X, Xie L, Wang G. Modulation of glucagon-like
peptide-1 release by berberine: in vivo and in vitro studies. Biochem Pharmacol.
Apr 1 2009;79(7):1000-1006.
17.) Al-masri IM, Mohammad MK, Tahaa MO. Inhibition of dipeptidyl peptidase
IV (DPP IV) is one of the mechanisms explaining the hypoglycemic effect of
berberine. J Enzyme Inhib Med Chem. Oct 2009;24(5):1061-1066.
18.) Yin J., Xing H., Ye J. Efficacy of Berberine in Patients with Type 2 Diabetes. Metabolism. 2008 May; 57(5): 712717.
19.) Abidi P, Chen W, Kraemer FB, Li H, Liu J. The medicinal plant goldenseal is a natural  LDL-lowering agent with multiple bioactive components and new action mechanisms.
J Lipid Res. 2006 Oct;47(10):2134-47. Epub 2006 Aug 2.
20.) Brusq JM, Ancellin N, Grondin P, Guillard R, Martin S, Saintillan Y, Issandou M.
Inhibition of lipid synthesis through activation of AMP kinase: an additional mechanism for the  hypolipidemic effects of berberine. J Lipid Res. 2006 Jun;47(6):1281-8. Epub 2006 Feb 28.
21.) Doggrell SA. Berberine–a novel approach to cholesterol lowering. Expert Opin Investig Drugs.  2005 May;14(5):683-5. [XXXanti-arrhythmic###]
22.) Kim WS, Lee YS, Cha SH, Jeong HW, Choe SS, Lee MR, Oh GT, Park HS, Lee KU, Lane MD,  Kim JB. Berberine improves lipid dysregulation in obesity by controlling central and peripheral AMPK  activity. Am J Physiol Endocrinol Metab. 2009 Apr;296(4):E812-9. Epub 2009 Jan 27.
23.)  Kong W, Wei J, Abidi P, Lin M, Inaba S, Li C, Wang Y, Wang Z, Si S, Pan H, Wang S, Wu J,  Wang Y, Li Z, Liu J, Jiang JD. Berberine is a novel cholesterol-lowering drug working through a  unique mechanism distinct from statins. Nat Med. 2004 Dec;10(12):1344-51. Epub 2004 Nov 7.
24.) Kong WJ, Wei J, Zuo ZY, Wang YM, Song DQ, You XF, Zhao LX, Pan HN, Jiang JD.
Combination of simvastatin with berberine improves the lipid-lowering efficacy. Metabolism. 2008  Aug;57(8):1029-37.
25.) Lee S, Lim HJ, Park JH, Lee KS, Jang Y, Park HY. Berberine-induced LDLR up-regulation  involves JNK pathway. Biochem Biophys Res Commun. 2007 Nov 3;362(4):853-7. Epub 2007 Aug  27.
26.) Li YH, Yang P, Kong WJ, Wang YX, Hu CQ, Zuo ZY, Wang YM, Gao H, Gao LM, Feng YC,  Du NN, Liu Y, Song DQ, Jiang JD. Berberine analogues as a novel class of the  low-density-lipoprotein receptor up-regulators: synthesis, structure-activity relationships, and  cholesterol-lowering efficacy. J Med Chem. 2009 Jan 22;52(2):492-501.
27.) Lau CW, Yao XQ, Chen ZY, Ko WH, Huang Y. Cardiovascular actions of berberine.
Cardiovasc Drug Rev. 2001 Fall;19(3):234-44.
28.) Li BX, Yang BF, Zhou J, Xu CQ, Li YR. Inhibitory effects of berberine on IK1, IK, and HERG  channels of cardiac myocytes. Acta Pharmacol Sin. 2001 Feb;22(2):125-31.
29.) Wang LH, Yu CH, Fu Y, Li Q, Sun YQ. Berberine elicits anti-arrhythmic effects via IK1/Kir2.1  in the rat type 2 diabetic myocardial infarction model. Phytother Res. 2011 Jan;25(1):33-7. doi:  10.1002/ptr.3097.
30.) Wang YX, Yao XJ, Tan YH. Effects of berberine on delayed afterdepolarizations in ventricular  muscles in vitro and in vivo. J Cardiovasc Pharmacol. 1994 May;23(5):716-22.
31.) Hong Y, Hui SS, Chan BT, Hou J. Effect of berberine on catecholamine levels in rats with  experimental cardiac hypertrophy. Life Sci. 2003 Apr 18;72(22):2499-507.
32.) Hong Y, Hui SC, Chan TY, Hou JY. Effect of berberine on regression of pressure-overload  induced cardiac hypertrophy in rats. Am J Chin Med. 2002;30(4):589-99.
33.) Marin-Neto JA, Maciel BC, Secches AL, Gallo Júnior L. Cardiovascular effects of berberine in  patients with severe congestive heart failure.Clin Cardiol. 1988 Apr;11(4):253-60.
34.) Zeng XH, Zeng XJ, Li YY. Efficacy and safety of berberine for congestive heart failure  secondary to ischemic or idiopathic dilated cardiomyopathy. Am J Cardiol. 2003 Jul 15;92(2):173-6.
35.) Chen TC, Lai KC, Yang JS, Liao CL, Hsia TC, Chen GW, Lin JJ, Lin HJ, Chiu TH, Tang YJ,  Chung JG. Involvement of reactive oxygen species and caspase-dependent pathway in  berberine-induced cell cycle arrest and apoptosis in C6 rat glioma cells. Int J Oncol. 2009  Jun;34(6):1681-90.
36.) Choi MS, Oh JH, Kim SM, Jung HY, Yoo HS, Lee YM, Moon DC, Han SB, Hong JT. Berberine  inhibits p53-dependent cell growth through induction of apoptosis of prostate cancer cells. Int J Oncol.  2009 May;34(5):1221-30.
37.) Eom KS, Kim HJ, So HS, Park R, Kim TY. Berberine-induced apoptosis in human glioblastoma T98G cells is mediated by endoplasmic reticulum stress accompanying reactive oxygen species and mitochondrial dysfunction. Biol Pharm Bull. 2010;33(10):1644-9.
38.) Eom KS, Hong JM, Youn MJ, So HS, Park R, Kim JM, Kim TY. Berberine induces G1 arrest and  apoptosis in human glioblastoma T98G cells through mitochondrial/caspases pathway. Biol Pharm Bull.  2008 Apr;31(4):558-62.
39.) Hamsa TP, Kuttan G. Berberine Inhibits Pulmonary Metastasis through Down-regulation of MMP in Metastatic B16F-10 Melanoma Cells. Phytother Res. 2011 Sep 26. doi: 10.1002/ptr.3586. [Epub ahead of print]
40.) Ho YT, Yang JS, Lu CC, Chiang JH, Li TC, Lin JJ, Lai KC, Liao CL, Lin JG, Chung JG. Berberine inhibits human tongue squamous carcinoma cancer tumor growth in a murine xenograft model. Phytomedicine. 2009 Sep;16(9):887-90. Epub 2009 Mar 20.
41.) Hsu WH, Hsieh YS, Kuo HC, Teng CY, Huang HI, Wang CJ, Yang SF, Liou YS, Kuo WH.
Berberine induces apoptosis in SW620 human colonic carcinoma cells through generation of reactive oxygen species and activation of JNK/p38 MAPK and FasL. Arch Toxicol. 2007 Oct;81(10):719-28. Epub 2007 Aug 3.
42.) Hwang JM, Kuo HC, Tseng TH, Liu JY, Chu CY. Berberine induces apoptosis through a  mitochondria/caspases pathway in human hepatoma cells. Arch Toxicol. 2006 Feb;80(2):62-73. Epub  2005 Sep 28.
43.) James MA, Fu H, Liu Y, Chen DR, You M. Dietary administration of berberine or Phellodendron amurense extract inhibits cell cycle progression and lung tumorigenesis. Mol Carcinog. 2011 Jan;50(1):1-7.
44.) Katiyar SK, Meeran SM, Katiyar N, Akhtar S. p53 Cooperates berberine-induced growth inhibition and apoptosis of non-small cell human lung cancer cells in vitro and tumor xenograft growth in vivo. Mol Carcinog. 2009 Jan;48(1):24-37.
45.) Kim S, Han J, Kim NY, Lee SK, Cho DH, Choi MY, Kim JS, Kim JH, Choe JH, Nam SJ, Lee  JE.Effect of berberine on p53 expression by TPA in breast cancer cells. Oncol Rep. 2012  Jan;27(1):210-5. doi: 10.3892/or.2011.1480. Epub 2011 Sep 29.
46.) Kim S, Choi JH, Kim JB, Nam SJ, Yang JH, Kim JH, Lee JE. Berberine suppresses  TNF-alpha-induced MMP-9 and cell invasion through inhibition of AP-1 activity in MDA-MB-231  human breast cancer cells. Molecules. 2008 Dec 3;13(12):2975-85.
47.) Li J, Cao B, Liu X, Fu X, Xiong Z, Chen L, Sartor O, Dong Y, Zhang H. Berberine suppresses  androgen receptor signaling in prostate cancer. Mol Cancer Ther. 2011 Aug;10(8):1346-56. Epub 2011  May 25.
48.) Lin CC, Kao ST, Chen GW, Ho HC, Chung JG. Apoptosis of human leukemia HL-60 cells and  murine leukemia WEHI-3 cells induced by berberine through the activation of caspase-3. Anticancer  Res. 2006 Jan-Feb;26(1A):227-42.
49.) Liu B, Wang G, Yang J, Pan X, Yang Z, Zang L. Berberine inhibits human hepatoma cell invasion  without cytotoxicity in healthy hepatocytes. PLoS One. 2011;6(6):e21416. Epub 2011 Jun 24.
50.) Liu Z, Liu Q, Xu B, Wu J, Guo C, Zhu F, Yang Q, Gao G, Gong Y, Shao C. Berberine induces p53-dependent cell cycle arrest and apoptosis of human osteosarcoma cells by inflicting DNA damage. Mutat Res. 2009 Mar 9;662(1-2):75-83. Epub 2008 Dec 31.
51.) Mantena SK, Sharma SD, Katiyar SK. Berberine, a natural product, induces G1-phase cell cycle  arrest and caspase-3-dependent apoptosis in human prostate carcinoma cells. Mol Cancer Ther. 2006  Feb;5(2):296-308.
52.) Mantena SK, Sharma SD, Katiyar SK. Berberine inhibits growth, induces G1 arrest and apoptosis  in human epidermoid carcinoma A431 cells by regulating Cdki-Cdk-cyclin cascade, disruption of  mitochondrial membrane potential and cleavage of caspase 3 and PARP. Carcinogenesis. 2006 Oct;  27(10):2018-27. Epub 2006 Apr 18.
53.) Meeran SM, Katiyar S, Katiyar SK. Berberine-induced apoptosis in human prostate cancer cells is  initiated by reactive oxygen species generation. Toxicol Appl Pharmacol. 2008 May 15;229(1):33-43.  Epub 2008 Jan 17.
54.) Patil JB, Kim J, Jayaprakasha GK. Berberine induces apoptosis in breast cancer cells (MCF-7) through mitochondrial-dependent pathway. Eur J Pharmacol. 2010 Oct 25;645(1-3):70-8. Epub 2010 Aug 4.
55.) Peng PL, Kuo WH, Tseng HC, Chou FP. Synergistic tumor-killing effect of radiation and berberine combined treatment in lung cancer: the contribution of autophagic cell death. Int J Radiat Oncol Biol Phys. 2008 Feb 1;70(2):529-42.
56.) Tan W, Li Y, Chen M, Wang Y. Berberine hydrochloride: anticancer activity and nanoparticulate  delivery system. Int J Nanomedicine. 2011;6:1773-7. Epub 2011 Aug 24.
57.) Yan K, Zhang C, Feng J, Hou L, Yan L, Zhou Z, Liu Z, Liu C, Fan Y, Zheng B, Xu Z. Induction  of G1 cell cycle arrest and apoptosis by berberine in bladder cancer cells. Eur J Pharmacol. 2011 Jul  1;661(1-3):1-7. Epub 2011 Apr 30.
58.) Yu FS, Yang JS, Lin HJ, Yu CS, Tan TW, Lin YT, Lin CC, Lu HF, Chung JG. Berberine inhibits WEHI-3 leukemia cells in vivo. In Vivo. 2007 Mar-Apr;21(2):407-12.
59.) Zhang RX, Dougherty DV, Rosenblum ML. Laboratory studies of berberine used alone and in combination with 1,3-bis(2-chloroethyl)-1-nitrosourea to treat malignant brain tumors. Chin Med J (Engl). 1990 Aug;103(8):658-65.
60.) Wei GX, Xu X, Wu CD. In vitro synergism between berberine and miconazole against planktonic and biofilm Candida cultures. Arch Oral Biol. 2011 Jun;56(6):565-72. Epub 2011 Jan 26.

Syntrax Matrix Whey – Vanilla

Whey Protein

Discontinued

Boost Immunity with Nature’s Superior Protein

Whey protein provides a biologically superior protein with natural immune factors, including lactoferrin and immunoglobulins.

In cancer medicine, whey has been found to offer “considerable protection to the host” over that of other types of protein, including soy, especially during chemotherapy and radiation. At low concentrations, whey inhibits the growth of breast cell cancer. Whey also protects cellular glutathione (a body-produced antioxidant) in normal cells during radiation therapy. This effect is not seen with other proteins.

For those with weak immune systems, or people who need to gain weight, whey is an easy way to get high quality protein and immune factors. For those on weight-loss diets, whey (such as a “Super Shake” below), makes a low calorie, low carb, high nutrient meal replacement. Try my recipe — it really tastes like an ice-cream milkshake!

Whey protein is beneficial for:

  • weight loss programs
  • weight and muscle gain programs
  • immune function
  • cancer
  • liver disease

My SuperShake recipe can be found here, and is a perfect way to get all of the benefits of whey and more!

Hot Chocolate

1 scoop chocolate whey
8 ounces hot water
1 scoop L-glutamine
(optional, for weight gain or GI repair)
To make
Heat water. Stir in whey powder. Drink. Enjoy.

Apple Pan Dowdy Pudding

2 scoops vanilla whey
2 tsps. Ez-Fiber
2 tsps. ground flax seed
Optional
1 scoop L-glutamine
1/8 cup chopped nuts
1 chopped apple
To make
Combine above ingredients. Add 8 ounces hot water. Stir. Put in cups. Enjoy hot or cold. Makes 2 servings.

Syntrax Vanilla Whey

This Whey Powder Is A Great Combination Of High Quality And Economical Price.

Sustained-Release Protein Blend
Undenatured Whey Protein, Micellar Casein & Egg Albumin
Vital Nutrition for Health & Recovery
Best Tasting Protein…Guaranteed
Glutamine Peptides
Mixes Instantly
Aspartame Free

Perfect For Low Carb Dieters!

SyntraxVanilla Whey
Product # 8050 (2.17 lbs or about 36 servings) $39.97

 

Syntrax Vanilla Whey

Nutrition Facts
Serving Size: 1 Level Scoop (30 g)
Servings per Container: 30
Amount Per Serving % Daily Value
Calories 110
Calories from Fat 15
Total Fat 1.5 g 2%
Saturated Fat 1 g 5%
Trans Fat 0 g
Cholesterol 40 mg 13%
Sodium 80 mg 3%
Potassium 200 mg 6%
Total Carbohydrates 2 g 1%
Dietary Fiber 0 g 0%
Sugars 2 g
Protein 23 g 46%
Vitamin A 0%
Vitamin C 0%
Calcium 15%
Iron 0%
Phosphorus 10%
Magnesium 6%
Important Amino Acids Per 100 Grams of Protein
Arginine 2.5 g *
Glutamine 8.4 g *
Histidine 2.1 g *
Isoleucine 5.8 g *
Leucine 10.3 g *
Lysine 8.7 g *
Methionine 2.2 g *
Phenylalanine 3.6 g *
Threonine 6.4 g *
Tryptophan 1.9 g *
Valine 6 g *
*Daily value not established.
Other Ingredients: Protein blend (whey protein concentrate, milk protein concentrate, egg albumin, hydrolyzed wheat gluten), natural and artificial flavors, soy lecithin, salt, acesulfame-K, sucralose.

 Syntrax Chocolate Whey

Nutrition Facts
Serving Size: 1 Level Scoop (32 g)
Servings per Container: 31
Amount Per Serving % Daily Value
Calories 120
Calories from Fat 20
Total Fat 2 g 3%
Saturated Fat 1 g 5%
Trans Fat 0 g
Cholesterol 40 mg 13%
Sodium 130 mg 5%
Potassium 280 mg 8%
Total Carbohydrates 3 g 1%
Dietary Fiber 1 g 4%
Sugars 2 g
Protein 23 g 46%
Vitamin A 0%
Vitamin C 0%
Calcium 15%
Iron 4%
Phosphorus 10%
Magnesium 8%
Important Amino Acids Per 100 Grams of Protein
Arginine 2.5 g *
Glutamine 8.4 g *
Histidine 2.1 g *
Isoleucine 5.8 g *
Leucine 10.3 g *
Lysine 8.7 g *
Methionine 2.2 g *
Phenylalanine 3.6 g *
Threonine 6.4 g *
Tryptophan 1.9 g *
Valine 6 g *
*Daily value not established.
Other Ingredients: Protein blend (whey protein concentrate, milk protein concentrate, egg albumin, hydrolyzed wheat gluten), dutch processed cocoa powder, natural and artificial flavors, soy lecithin, salt, acesulfame-K, sucralose.

 

Fertility Restore Maxi Marine O-3 (120 softgels)

Maxi Marine O-3

High Potency Enteric Coated Ultra-Pure Essential FattyAcids From Norwegian Arctic-Harvested Fish

Maxi Marine O-3 is an ultra-pure, ultra-high potency fish oil from anchovies and sardines harvested in pristine Norwegian Arctic waters. Purity is further assured by molecular distillation and every batch is verified by an independent third-party lab to be free of PCB’s, heavy metals and pesticides. Vitamin E (mixed tocopherols) is added to maintain maximum freshness and enteric coating ensures no “fishy” after-taste.

EPA and DHA, the “active ingredients” in fish oil, are long-chain Omega-3 Essential Fatty Acids that differ from short-chain Omega-3’s found in plant sources. EPA and DHA are beneficial for Cardiovascular (Heart) Health, Alzheimer’s Disease and other Dementias, Mental Health, Child Development, Attention-Deficit Disorders and Social/Cognitive Development, Diabetes, Inflammatory Diseases (rheumatoid arthritis, IBS/Crohn’s, asthma, infertility, etc.) and Cancer.

EPA/DHA (which occur together in fish oils) have numerous studies supporting their anti-inflammatory properties and use in multiple hyper-inflammatory and autoimmune states. (14-24) EPA and DHA decrease NK cell activity (14,24-29), and this effect is synergistic when both EPA and DHA are used together.(18,28). EPA, but not ALA, also decreases TNF-alpha.(23-25).

While ALA (flax oil) has some similar benefits, it must be taken in significantly higher doses because ultimately the anti-inflammatory effect is seen when ALA is converted to EPA, as follows:

ALA (flax oil) >>> SDA (stearidonic acid) >>> EPA (fish oil) >>> Prostaglandin E3

An estimated 0.07% of ALA is converted to EPA in healthy people (30,31), but the conversion rate may be less when the delta-6-desaturase (d6d) enzyme is under functioning or when there is high competition from dietary Omega-6’s (both use the d6d enzyme). Assuming that converting enzymes are normal in a fertility clinic patient, a dose comparison would be 14 grams of flax oil vs. 1 grams of fish oil.

Studies have shown that ALA increases EPA but not DHA.(31) Some studies have shown that ALA does not reduce inflammatory markers (23,29). For these reasons, fish oil is preferable to flax oil for raising EPA/DHA levels (33).

Suggested Use: Adults take one or two capsules daily with meals or more if directed by your health care provider. Please note that the EPA/DHA content of Maxi Marine O-3 (700mg total) is two and a half times more concentrated than most fish oils.

Marine Lipid Concentrate: 1000mg – which provides:
EPA (eicosapentaenoic acid) 420mg
DHA (docosahexaenoic acid) 280mg

Because Maxi Marine is so concentrated it is a much more economical choice for those using higher doses of these important oils. Our regular brand of Ultra-Pure Fish Oil costs approximately 67 cents per gram while our higher-potency Maxi Marine O-3 is only 58 cents per gram. Higher potency means fewer capsules per day, representing significant cost savings.

Use in Fertility Restoration therapy is normally one capsule twice daily.

STORAGE: Store in a cool place and keep out of reach of children.

Contains No wheat, gluten, corn protein, yeast, dairy, artificial colors, sweeteners or preservatives.

Product # F310 – Maxi Marine O3 – 120 softgels – $54.95

Enter Quantity Desired and Click “Add To Cart” Button

 

References:

14.) Mukaro VR, Costabile M, Murphy KJ, Hii CS, Howe PR, Ferrante A. Leukocyte numbers and function in subjects eating n-3 enriched foods: selective depression of natural killer cell levels. Arthritis Res Ther. 2008;10(3):R57. Epub 2008 May 14.
CONCLUSION: The data show that regular long-term consumption of n-3 enriched foods leads to lower numbers of NK cells and neutrophil iodination activity but higher lymphotoxin production by lymphocytes. These changes are consistent with decreased inflammatory reaction and
tissue damage seen in patients with inflammatory disorders receiving n-3 LCPUFA supplementation.

15.) Calder PC. Dietary modification of inflammation with lipids. Proc Nutr Soc. 2002 Aug;61(3):345-58. CONCLUSION: EPA is anti-inflammatory

16.) Calder PC. N-3 polyunsaturated fatty acids and inflammation: from molecular biology to the clinic. Lipids. 2003 Apr;38(4):343-52.
CONCLUSION: EPA is anti-inflammatory

17.) Simopoulos AP. Omega-3 fatty acids in inflammation and autoimmune diseases. J Am Coll Nutr. 2002 Dec;21(6):495-505.
Conclusion: Fish oil more biologically active than ALA; anti-inflammatory properties; useful in RA and MS among others

18.) Calder PC. Session 3: Joint Nutrition Society and Irish Nutrition and Dietetic Institute Symposium on ‘Nutrition and autoimmune disease’ PUFA, inflammatory processes and rheumatoid arthritis. Proc Nutr Soc. 2008 Nov;67(4):409-18.
Conclusion: fish oil (both EPA and DHA) are anti-inflammatory

19.) Rupp H, Wagner D, Rupp T, Schulte LM, Maisch B. Risk stratification by the “EPA+DHA level” and the “EPA/AA ratio” focus on anti-inflammatory and antiarrhythmogenic effects of long-chain omega-3 fatty acids. Herz. 2004 Nov;29(7):673-85.
Conclusion: DOSE:2-4 g/day of 84% EPA+DHA ethyl esters for anti-inflammatory effects

20.) Cleland LG, Caughey GE, James MJ, Proudman SM. Reduction of cardiovascular risk factors with longterm fish oil treatment in early rheumatoid arthritis.J Rheumatol. 2006 Oct;33(10):1973-9. Epub 2006 Aug 1.
Conclusion: anti-inflammatory effects of fish oil in RA patients; also reduced NSAID use with fish oil

21.) Cleland LG, James MJ, Proudman SM. The role of fish oils in the treatment of rheumatoid arthritis. Drugs. 2003;63(9):845-53.
Conclusion: fish oil anti-inflammatory in RA

22.) Kremer JM, Lawrence DA, Petrillo GF, Litts LL, Mullaly PM, Rynes RI, Stocker RP, Parhami N, Greenstein NS, Fuchs BR, et al. Effects of high-dose fish oil on rheumatoid arthritis after stopping nonsteroidal antiinflammatory drugs. Clinical and immune correlates. Arthritis
Rheum. 1995 Aug;38(8):1107-14.
Conclusion: Fish oil anti-inflammatory in RA

23.) Pischon T, Hankinson SE, Hotamisligil GS, Rifai N, Willett WC, Rimm EB. Habitual dietary intake of n-3 and n-6 fatty acids in relation to inflammatory markers among US men and women. Circulation. 2003 Jul 15;108(2):155-60. Epub 2003 Jun 23.
Conclusion: Fish oil but not ALA associated with decreased inflammatory markers; Fish oil decreased TNF

24.) Ferrucci L, Cherubini A, Bandinelli S, Bartali B, Corsi A, Lauretani F, Martin A, Andres-Lacueva C, Senin U, Guralnik JM. Relationship of plasma polyunsaturated fatty acids to circulating inflammatory markers. J Clin Endocrinol Metab. 2006 Feb;91(2):439-46. Epub 2005 Oct 18.
Conclusion: Higher EFA’s accociated with lower inflammatory markers; n-3 lowers TNF-alpha and inflammation

25.) Sundrarjun T, Komindr S, Archararit N, Dahlan W, Puchaiwatananon O, Angthararak S, Udomsuppayakul U, Chuncharunee S. Effects of n-3 fatty acids on serum interleukin-6, tumour necrosis factor-alpha and soluble tumour necrosis factor receptor p55 in active rheumatoid
arthritis. J Int Med Res. 2004 Sep-Oct;32(5):443-54.
Conclusion: Fish oil decreaases TNF-alpha, soluble tumour necrosis factor receptor p55 (sTNF-R p55), CPR and I-6 in RA patients

26.) Yamashita N, Sugiyama E, Hamazaki T, Yano S.Inhibition of natural killer cell activity by eicosapentaenoic acid in vivo and in vitro.Biochem Biophys Res Commun. 1988 Jan 15;150(1):497-505.
Conclusion: EPA decreases NK activity

27.) Yamashita N, Yokoyama A, Hamazaki T, Yano S. Inhibition of natural killer cell activity of human lymphocytes by eicosapentaenoic acid. Biochem Biophys Res Commun. 1986 Aug 14;138(3):1058-67.
Conclusion: EPA decreases NK activity

28) Yamashita N, Maruyama M, Yamazaki K, Hamazaki T, Yano S. Effect of eicosapentaenoic and docosahexaenoic acid on natural killer cell activity in human peripheral blood lymphocytes. Clin Immunol Immunopathol. 1991 Jun;59(3):335-45.
Conclusion: EPA and DHA both inhibit NK cell activity; the effect is greater (synergistic) when both are used together

29.) Thies F, Nebe-von-Caron G, Powell JR, Yaqoob P, Newsholme EA, Calder PC. Dietary supplementation with eicosapentaenoic acid, but not with other long-chain n-3 or n-6 polyunsaturated fatty acids, decreases natural killer cell activity in healthy subjects aged >55 y. Am J Clin Nutr. 2001 Mar;73(3):539-48.
Conclusion: NK cell activity was not significantly affected by the placebo, ALA, GLA, AA, or DHA treatment. Fish oil caused a significant reduction (mean decline: 48%) in NK cell activity that was fully reversed by 4 wk after supplementation had ceased. CONCLUSION: A moderate amount of EPA but not of other n-6 or n-3 polyunsaturated fatty acids can decrease NK cell activity in healthy subjects.” DOSE: 1 g EPA plus DHA (720 mg EPA + 280 mg DHA) daily

30.) James MJ, Ursin VM, Cleland LG. Metabolism of stearidonic acid in human subjects: comparison with the metabolism of other n-3 fatty acids. Am J Clin Nutr. 2003 May;77(5):1140-5.
Conclusion: increases of EPA from various sources was 1:0.3:0.07 for EPA:SDA:ALA.

31.) Wallace FA, Miles EA, Calder PC. Comparison of the effects of linseed oil and different doses of fish oil on mononuclear cell function in healthy human subjects. Br J Nutr. 2003 May;89(5):679-89.
Conclusion: ALA increases EPA but not DHA; higher dose needed

32.) Arterburn LM, Hall EB, Oken H. Distribution, interconversion, and dose response of n-3 fatty acids in humans. Am J Clin Nutr. 2006 Jun;83(6 Suppl):1467S-1476S.
Conclusion: ALA not well-converted to EPA and DHA; DOSE: DHA 2 g/day for maximal tissue response

33.) Young GS, Conquer JA, Thomas R. Effect of randomized supplementation with high dose olive, flax or fish oil on serum phospholipid fatty acid levels in adults with attention deficit hyperactivity disorder. Reprod Nutr Dev. 2005 Sep-Oct;45(5):549-58.
Conclusion: fish oil prefferable to flax oil for raising EPA/DHA levels and decreasing AA/EPA ratios

 

Fertility Restore Maxi Flavone 60 caps

Maxi Marine O-3

High Potency Enteric Coated Ultra-Pure Essential FattyAcids From Norwegian Arctic-Harvested Fish

Maxi Marine O-3 is an ultra-pure, ultra-high potency fish oil from anchovies and sardines harvested in pristine Norwegian Arctic waters. Purity is further assured by molecular distillation and every batch is verified by an independent third-party lab to be free of PCB’s, heavy metals and pesticides. Vitamin E (mixed tocopherols) is added to maintain maximum freshness and enteric coating ensures no “fishy” after-taste.

EPA and DHA, the “active ingredients” in fish oil, are long-chain Omega-3 Essential Fatty Acids that differ from short-chain Omega-3’s found in plant sources. EPA and DHA are beneficial for Cardiovascular (Heart) Health, Alzheimer’s Disease and other Dementias, Mental Health, Child Development, Attention-Deficit Disorders and Social/Cognitive Development, Diabetes, Inflammatory Diseases (rheumatoid arthritis, IBS/Crohn’s, asthma, infertility, etc.) and Cancer.

EPA/DHA (which occur together in fish oils) have numerous studies supporting their anti-inflammatory properties and use in multiple hyper-inflammatory and autoimmune states. (14-24) EPA and DHA decrease NK cell activity (14,24-29), and this effect is synergistic when both EPA and DHA are used together.(18,28). EPA, but not ALA, also decreases TNF-alpha.(23-25).

While ALA (flax oil) has some similar benefits, it must be taken in significantly higher doses because ultimately the anti-inflammatory effect is seen when ALA is converted to EPA, as follows:

ALA (flax oil) >>> SDA (stearidonic acid) >>> EPA (fish oil) >>> Prostaglandin E3

An estimated 0.07% of ALA is converted to EPA in healthy people (30,31), but the conversion rate may be less when the delta-6-desaturase (d6d) enzyme is under functioning or when there is high competition from dietary Omega-6’s (both use the d6d enzyme). Assuming that converting enzymes are normal in a fertility clinic patient, a dose comparison would be 14 grams of flax oil vs. 1 grams of fish oil.

Studies have shown that ALA increases EPA but not DHA.(31) Some studies have shown that ALA does not reduce inflammatory markers (23,29). For these reasons, fish oil is preferable to flax oil for raising EPA/DHA levels (33).

Suggested Use: Adults take one or two capsules daily with meals or more if directed by your health care provider. Please note that the EPA/DHA content of Maxi Marine O-3 (700mg total) is two and a half times more concentrated than most fish oils.

Marine Lipid Concentrate: 1000mg – which provides:
EPA (eicosapentaenoic acid) 420mg
DHA (docosahexaenoic acid) 280mg

Because Maxi Marine is so concentrated it is a much more economical choice for those using higher doses of these important oils. Our regular brand of Ultra-Pure Fish Oil costs approximately 67 cents per gram while our higher-potency Maxi Marine O-3 is only 58 cents per gram. Higher potency means fewer capsules per day, representing significant cost savings.

Use in Fertility Restoration therapy is normally one capsule twice daily.

STORAGE: Store in a cool place and keep out of reach of children.

Contains No wheat, gluten, corn protein, yeast, dairy, artificial colors, sweeteners or preservatives.

Product # F310 – Maxi Marine O3 – 120 softgels – $54.95

Enter Quantity Desired and Click “Add To Cart” Button

 

References:

14.) Mukaro VR, Costabile M, Murphy KJ, Hii CS, Howe PR, Ferrante A. Leukocyte numbers and function in subjects eating n-3 enriched foods: selective depression of natural killer cell levels. Arthritis Res Ther. 2008;10(3):R57. Epub 2008 May 14.
CONCLUSION: The data show that regular long-term consumption of n-3 enriched foods leads to lower numbers of NK cells and neutrophil iodination activity but higher lymphotoxin production by lymphocytes. These changes are consistent with decreased inflammatory reaction and
tissue damage seen in patients with inflammatory disorders receiving n-3 LCPUFA supplementation.

15.) Calder PC. Dietary modification of inflammation with lipids. Proc Nutr Soc. 2002 Aug;61(3):345-58. CONCLUSION: EPA is anti-inflammatory

16.) Calder PC. N-3 polyunsaturated fatty acids and inflammation: from molecular biology to the clinic. Lipids. 2003 Apr;38(4):343-52.
CONCLUSION: EPA is anti-inflammatory

17.) Simopoulos AP. Omega-3 fatty acids in inflammation and autoimmune diseases. J Am Coll Nutr. 2002 Dec;21(6):495-505.
Conclusion: Fish oil more biologically active than ALA; anti-inflammatory properties; useful in RA and MS among others

18.) Calder PC. Session 3: Joint Nutrition Society and Irish Nutrition and Dietetic Institute Symposium on ‘Nutrition and autoimmune disease’ PUFA, inflammatory processes and rheumatoid arthritis. Proc Nutr Soc. 2008 Nov;67(4):409-18.
Conclusion: fish oil (both EPA and DHA) are anti-inflammatory

19.) Rupp H, Wagner D, Rupp T, Schulte LM, Maisch B. Risk stratification by the “EPA+DHA level” and the “EPA/AA ratio” focus on anti-inflammatory and antiarrhythmogenic effects of long-chain omega-3 fatty acids. Herz. 2004 Nov;29(7):673-85.
Conclusion: DOSE:2-4 g/day of 84% EPA+DHA ethyl esters for anti-inflammatory effects

20.) Cleland LG, Caughey GE, James MJ, Proudman SM. Reduction of cardiovascular risk factors with longterm fish oil treatment in early rheumatoid arthritis.J Rheumatol. 2006 Oct;33(10):1973-9. Epub 2006 Aug 1.
Conclusion: anti-inflammatory effects of fish oil in RA patients; also reduced NSAID use with fish oil

21.) Cleland LG, James MJ, Proudman SM. The role of fish oils in the treatment of rheumatoid arthritis. Drugs. 2003;63(9):845-53.
Conclusion: fish oil anti-inflammatory in RA

22.) Kremer JM, Lawrence DA, Petrillo GF, Litts LL, Mullaly PM, Rynes RI, Stocker RP, Parhami N, Greenstein NS, Fuchs BR, et al. Effects of high-dose fish oil on rheumatoid arthritis after stopping nonsteroidal antiinflammatory drugs. Clinical and immune correlates. Arthritis
Rheum. 1995 Aug;38(8):1107-14.
Conclusion: Fish oil anti-inflammatory in RA

23.) Pischon T, Hankinson SE, Hotamisligil GS, Rifai N, Willett WC, Rimm EB. Habitual dietary intake of n-3 and n-6 fatty acids in relation to inflammatory markers among US men and women. Circulation. 2003 Jul 15;108(2):155-60. Epub 2003 Jun 23.
Conclusion: Fish oil but not ALA associated with decreased inflammatory markers; Fish oil decreased TNF

24.) Ferrucci L, Cherubini A, Bandinelli S, Bartali B, Corsi A, Lauretani F, Martin A, Andres-Lacueva C, Senin U, Guralnik JM. Relationship of plasma polyunsaturated fatty acids to circulating inflammatory markers. J Clin Endocrinol Metab. 2006 Feb;91(2):439-46. Epub 2005 Oct 18.
Conclusion: Higher EFA’s accociated with lower inflammatory markers; n-3 lowers TNF-alpha and inflammation

25.) Sundrarjun T, Komindr S, Archararit N, Dahlan W, Puchaiwatananon O, Angthararak S, Udomsuppayakul U, Chuncharunee S. Effects of n-3 fatty acids on serum interleukin-6, tumour necrosis factor-alpha and soluble tumour necrosis factor receptor p55 in active rheumatoid
arthritis. J Int Med Res. 2004 Sep-Oct;32(5):443-54.
Conclusion: Fish oil decreaases TNF-alpha, soluble tumour necrosis factor receptor p55 (sTNF-R p55), CPR and I-6 in RA patients

26.) Yamashita N, Sugiyama E, Hamazaki T, Yano S.Inhibition of natural killer cell activity by eicosapentaenoic acid in vivo and in vitro.Biochem Biophys Res Commun. 1988 Jan 15;150(1):497-505.
Conclusion: EPA decreases NK activity

27.) Yamashita N, Yokoyama A, Hamazaki T, Yano S. Inhibition of natural killer cell activity of human lymphocytes by eicosapentaenoic acid. Biochem Biophys Res Commun. 1986 Aug 14;138(3):1058-67.
Conclusion: EPA decreases NK activity

28) Yamashita N, Maruyama M, Yamazaki K, Hamazaki T, Yano S. Effect of eicosapentaenoic and docosahexaenoic acid on natural killer cell activity in human peripheral blood lymphocytes. Clin Immunol Immunopathol. 1991 Jun;59(3):335-45.
Conclusion: EPA and DHA both inhibit NK cell activity; the effect is greater (synergistic) when both are used together

29.) Thies F, Nebe-von-Caron G, Powell JR, Yaqoob P, Newsholme EA, Calder PC. Dietary supplementation with eicosapentaenoic acid, but not with other long-chain n-3 or n-6 polyunsaturated fatty acids, decreases natural killer cell activity in healthy subjects aged >55 y. Am J Clin Nutr. 2001 Mar;73(3):539-48.
Conclusion: NK cell activity was not significantly affected by the placebo, ALA, GLA, AA, or DHA treatment. Fish oil caused a significant reduction (mean decline: 48%) in NK cell activity that was fully reversed by 4 wk after supplementation had ceased. CONCLUSION: A moderate amount of EPA but not of other n-6 or n-3 polyunsaturated fatty acids can decrease NK cell activity in healthy subjects.” DOSE: 1 g EPA plus DHA (720 mg EPA + 280 mg DHA) daily

30.) James MJ, Ursin VM, Cleland LG. Metabolism of stearidonic acid in human subjects: comparison with the metabolism of other n-3 fatty acids. Am J Clin Nutr. 2003 May;77(5):1140-5.
Conclusion: increases of EPA from various sources was 1:0.3:0.07 for EPA:SDA:ALA.

31.) Wallace FA, Miles EA, Calder PC. Comparison of the effects of linseed oil and different doses of fish oil on mononuclear cell function in healthy human subjects. Br J Nutr. 2003 May;89(5):679-89.
Conclusion: ALA increases EPA but not DHA; higher dose needed

32.) Arterburn LM, Hall EB, Oken H. Distribution, interconversion, and dose response of n-3 fatty acids in humans. Am J Clin Nutr. 2006 Jun;83(6 Suppl):1467S-1476S.
Conclusion: ALA not well-converted to EPA and DHA; DOSE: DHA 2 g/day for maximal tissue response

33.) Young GS, Conquer JA, Thomas R. Effect of randomized supplementation with high dose olive, flax or fish oil on serum phospholipid fatty acid levels in adults with attention deficit hyperactivity disorder. Reprod Nutr Dev. 2005 Sep-Oct;45(5):549-58.
Conclusion: fish oil prefferable to flax oil for raising EPA/DHA levels and decreasing AA/EPA ratios

Fertility Restore Lycopene 15mg (30 Softgel Capsules)

LYCOPENE

Prostate Protection from Tomatoes

Lycopene, a concentrated carotene found in tomatoes, is the most effective free radical neutralizer in the carotenoid family. Studies have shown that men who eat tomato products (especially tomato paste, which is high in lycopene), have a lower incidence of prostate cancer.(2,3) In fact, the heat-processing of tomatoes actually makes the lycopene easier to use in the body.(1)

Today, high potency lycopene is used in the holistic treatment of prostate cancer as well as prevention.

Suggested dose: 1 capsule per day.

Each (one) capsule contains: Lyocopene (from tomatoes) 15 mg

Product # 166 – Lyocopene 15 mg (30 Capsules)$15.95

Enter Quantity Desired and Click “Add To Cart” Button

 

References

1.) Gartner C, Stahl W, Sies H. Lycopene is more bioavailable from tomato paste than from fresh tomatoes. Am J Clin Nutr 1997;66:116-22.
2.) Gann PH, Ma J, Giovannucci E, et al. Lower prostate cancer risk in men with elevated plasma lycopene levels: results of a prospective analysis. Cancer Res 1999;59:1225-30.
3.) Clinton SK, Emenhiser C, Schwartz SJ, et al. Cis-trans lycopene isomers, carotenoids, and retinol in the human prostate. Cancer Epidemiol Biomarkers Prev 1996;5:823-33

 

Food Intolerance Profile – Vegetarian

FOOD INTOLERANCE PANEL

Food allergies are a common, undiagnosed cause of many health problems. This test screens for antibodies against 96 different foods commonly found in a typical American diet. (95 foods in the vegetarian panel). The test provides the same clinical results that can be obtained from a local laboratory without the added expense and inconvenience because you collect the blood specimen yourself using a simple finger-stick method.

The kit includes all collection materials and the means to ship the specimen to the lab free of charge. Results are provided within 10-14 days from the time the test was received at the lab. Each patient report comes complete with a customized 4-Day Elimination/Rotation Diet Guideline and an easy-to-read food family booklet.

Due to state regulations, this test is not available to New York and New Jersey residents.

GENERAL FOOD INTOLERANCE PANEL #985 $265.00

VEGETARIAN FOOD INTOLERANCE PANEL #987 $276.00

Food Intolerance Profile (Standard)

FOOD INTOLERANCE PANEL

Food allergies are a common, undiagnosed cause of many health problems. This test screens for antibodies against 96 different foods commonly found in a typical American diet. (95 foods in the vegetarian panel). The test provides the same clinical results that can be obtained from a local laboratory without the added expense and inconvenience because you collect the blood specimen yourself using a simple finger-stick method.

The kit includes all collection materials and the means to ship the specimen to the lab free of charge. Results are provided within 10-14 days from the time the test was received at the lab. Each patient report comes complete with a customized 4-Day Elimination/Rotation Diet Guideline and an easy-to-read food family booklet.

Due to state regulations, this test is not available to New York and New Jersey residents.

GENERAL FOOD INTOLERANCE PANEL #985 $265.00

VEGETARIAN FOOD INTOLERANCE PANEL #987 $276.00

Coconut Oil (Organic, Virgin) 15 fl. ounces


This Oil Should Be in Your Kitchen AND Your Medicine Cabinet

Coconut oil is a saturated fat, which means it doesn’t go rancid when heated or when stored for long periods of time.

Essential Fatty Acids (EFA’s) turn into unhealthy “trans fats” when heated, so although you need EFA’s for good health, you should NOT cook with them! Even olive oil, a polyunsaturated fat (PUFA)  should NOT be used for cooking. (Olive oil is NOT an essential fat).

Coconut oil and other saturated fats are heat-stable. For frying and deep-frying, coconut oil is one of the very best oils to use.

In addition to being heat-stable and great for cooking, coconut oil has the following health advantages:

  • Coconut oil contains lauric acid and other a medium-chain triglycerides (MCT’s) with powerful antimicrobial effects against a wide range of bacteria, viruses, fungi / yeasts and protozoa. (1-7)
  • The MCT’s in coconut oil activate the immune system. (8,9)
  • Coconut oil’s MCT’s have proven anti-tumor effects. (10,11)
  • MCT’s in coconut oil aid weight loss three ways: by decreasing hunger, increasing fat-burning and increasing metabolism. (12-19)
  • Lowers cholesterol and improves blood fat levels. (20-22)

If you’ve heard bad things about saturated fats, you should know that it’s all nonsense – bogus B.S. promoted by the soy oil industry and Big Business. (Can you say “Proctor and Gamble”?) Read about the unjustified “bad press” that this miraculous oil has suffered here: Saturated Fat: Another Big, Fat Lie

We offer organic, virgin coconut oil, the finest quality available. Use coconut to cook, make pie crusts or anything that calls for “Crisco” (Crystallized Cottonseed Oil, a product not fit for human consumption).

Use Coconut Oil, two to four tablespoons per day as a food, substituted for whatever oils you currently cook with (except butter which is a true health food). Heck, you can even use coconut oil on your skin as a moisturizer, protectant and anti-microbial!

Coconut Oil (Organic, Virgin) 15 fl. ounces   $15.95

Please Note: Coconut oil is slippery stuff – and it may seep from it’s container during shipment. We have received reports of coconut oil seeping out of still-tightly-sealed jars! We double-bag this product and pack it carefully for shipping but we cannot be responsible for leakage during shipment – there will be no returns or refunds on this product.

References

1.) Antimicrobial activity of potassium hydroxide and lauric acid against microorganisms associated with poultry processing. J Food Prot. 2006 Jul;69(7):1611-5.
2.) In vitro activity of lauric acid or myristylamine in combination with six antimicrobial agents against methicillin-resistant Staphylococcus aureus (MRSA). Int J Antimicrob Agents. 2006 Jan;27(1):51-7. Epub 2005 Nov 28.
3.) Susceptibility of Clostridium perfringens to C-C fatty acids.Lett Appl Microbiol. 2005;41(1):77-81.
4.) Effect of lauric acid and nisin-impregnated soy-based films on the growth of Listeria monocytogenes on turkey bologna. Poult Sci. 2002 May;81(5):721-6.
5.) Inhibition of bacterial foodborne pathogens by the lactoperoxidase system in combination with monolaurin. Int J Food Microbiol. 2002 Feb 25;73(1):1-9.
6.) Fatty acids and derivatives as antimicrobial agents. Antimicrob Agents Chemother. 1972 Jul;2(1):23-8.
7.) Susceptibility of Helicobacter pylori to bactericidal properties of medium-chain monoglycerides and free fatty acids. Antimicrob Agents Chemother. 1996 Feb;40(2):302-6.
8.) Saturated triglycerides and fatty acids activate neutrophils depending on carbon chain-length. Eur J Clin Invest. 2002 Apr;32(4):285-9.
9.) Immunonutrition–supplementary amino acids and fatty acids ameliorate immune deficiency in critically ill patients. Langenbecks Arch Surg. 2001 Aug;386(5):369-76.
10.) Antitumor effect of medium-chain triglyceride and its influence on the self-defense system of the body. Cancer Detect Prev. 1998;22(3):219-24.
11.) Effects of calcitriol, seocalcitol, and medium-chain triglyceride on a canine transitional cell carcinoma cell line. Anticancer Res. 2005 Jul-Aug;25(4):2689-96.
12.) Value of VLCD supplementation with medium chain triglycerides.Int J Obes Relat Metab Disord. 2001 Sep;25(9):1393-400.
13.) The thermic effect is greater for structured medium- and long-chain triacylglycerols versus long-chain triacylglycerols in healthy young women. Metabolism. 2001 Jan;50(1):125-30.
14.) Greater rise in fat oxidation with medium-chain triglyceride consumption relative to long-chain triglyceride is associated with lower initial body weight and greater loss of subcutaneous adipose tissue. Int J Obes Relat Metab Disord. 2003 Dec;27(12):1565-71.
15.) Thermogenesis in humans during overfeeding with medium-chain triglycerides.Metabolism. 1989 Jul;38(7):641-8.
16.) Dietary medium-chain triacylglycerols suppress accumulation of body fat in a double-blind, controlled trial in healthy men and women.J Nutr. 2001 Nov;131(11):2853-9.
17.) Enhanced thermogenesis and diminished deposition of fat in response to overfeeding with diet containing medium chain triglyceride. Am J Clin Nutr. 1982 Apr;35(4):678-82.
18.) Medium-chain triglycerides increase energy expenditure and decrease adiposity in overweight men.Obes Res. 2003 Mar;11(3):395-402.
19.) Comparison of diet-induced thermogenesis of foods containing medium- versus long-chain triacylglycerols.J Nutr Sci Vitaminol (Tokyo). 2002 Dec;48(6):536-40.
20.) Effect of dietary medium- and long-chain triacylglycerols (MLCT) on accumulation of body fat in healthy humans. Asia Pac J Clin Nutr. 2003;12(2):151-60.
21.) Beneficial effects of virgin coconut oil on lipid parameters and in vitro LDL oxidation. Clin Biochem. 2004 Sep;37(9):830-5.
22.) Effect of medium-chain triglycerides on the postprandial triglyceride concentration in healthy men. Biosci Biotechnol Biochem. 2003 Jan;67(1):46-53.

Heavy Metal / Urine Chelation Test

Heavy Metal / Urine Chelation Test.

The most accurate test for heavy metals in the body.

Heavy metals — including mercury, lead, cadmium, iron and arsenic — can be an overlooked cause of high blood pressure, neurological diseases and symptoms, chronic fatigue and other complaints for which there is no clear diagnosis.

Blood tests for heavy metals are virtually useless because the body clears toxic metals from the bloodstream very rapidly and stores them in tissues such as bone and metabolically active tissues where they will cause less damage.

Hair mineral analysis for heavy metals is more accurate than blood testing but is subject to “false negatives.” If one or more metals are found to be high based on a hair mineral analysis, there is definitely a toxic mineral problem. But if the hair test comes back negative, that doesn’t prove that you’re free from heavy metal toxicity. The metal can be stored in other tissues in the body, not the hair.

The best test for the presence of heavy metals is a chelation test. This test uses a “chelating agent” (EDTA, DMSA) to pull metals out of storage sites where they are then eliminated in the urine. Older versions of the test required in-office IV chelation prior to testing.

Fortunately, there is a newer oral chelation challenge / urine test that many holistic physicians feel is just as accurate as IV chelation testing, at a fraction of the cost and inconvenience.

The test entails fasting for several hours, then taking an oral chelating agent called DMSA (Dimercaptosuccinic Acid), a potent chelating agent. The urine is then collected for the next 6 hours and a send a sample of this to the lab in a pre-paid mailer.

This test kit includes DMSA sufficient to complete the test

Heavy Metal / Urine Chelation Test # N267 $279