Dr. Myatt’s Cardiovascular Risk Checklist

A Medical White Paper Presented By Dr. Dana Myatt

February is “Heart Month.” Here’s Your Heart-Risk Checklist.

Shocking Facts about Heart Attacks

February is heart month, and in honor of your heart, I have prepared a special heart-risk assessment and report for you. First, some surprising statistics about heart disease. These “fast facts” will help you know why my heart-risk checklist is so potentially important.

Heart disease is the #1 cause of death in the US.1 2,200 people die every day from heart disease.

As many as 50% of all people who have a heart attack do not have ANY classic risk factors, although one study argues that this number is actually only 20%. “Only” a 20% chance of having a heart attack with no known risk factors? I don’t know about you, but that still sounds like a big risk to me.

As many as fifty percent (50%) of all first heart attacks are last heart attacks if you get my drift. Half of all people who have a heart attack die from “sudden cardiac death.” No second chances. No “jump-starting” the heart with a defibrillator. No bypass surgery or stents. Just gone in a heartbeat.

People with NO conventional risk factors are more likely to die “sudden death” from a first heart attack. Sudden cardiac death is the first and only sign of heart disease in this group.

You could be a non-smoker with a normal body weight, total cholesterol below 200, LDL below 100, HDL above 50. You don’t smoke, are not diabetic and have no family history of heart disease. Good for you. You doctor has just given you a clean bill of health and told you your heart is fine. And you could die of a heart attack as you leave the doctor’s office. Remember, twenty to fifty percent of all people who have a heart attack do not have ANY conventionally-tested heart risks.

Emerging Risk Factors: The “Other Risks” No One Is Telling You About

Routinely screened conventional risk factors include blood fats (total cholesterol, LDL, HDL), blood pressure, smoking, and diabetes.4 Additional testing might include a cardiac stress test (the “treadmill test”). Overweight/obesity, family history and activity levels should also be considered.

Unfortunately, 20-50% of people who have heart attacks are “normal” for all of these tests and markers. It’s the folks with “all normal” risk factors who have the greatest likelihood of having a fatal heart attack.

Conventional medicine acknowledges that there are a number of other risk factors for heart disease. These are called “emerging risk factors” because the information is still “emerging” or coming to light.

Unfortunately, tests for these “emerging risk factors” are not yet ordered by most conventional physicians nor are they typically covered by insurance. Many of them will be “standard of care” in conventional medicine some day in the future. Will “some day” be soon enough for you or me?

Good News About “Emerging Risk Factors”

The good news is many of the most important of these “other risks” can be tested at an affordable price. They are not obscure tests with thousand-dollar price tags.

The OTHER good news is that there are safe, natural, proven options for correcting abnormalities if and when they are found. After all, what good would it be to know about a risk factor if there was nothing you could do about it?

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Please print this Medical White Paper, including the pages of references, so that you can show it to your doctor / cardiologist. When he / she tells you that 1) he has never heard of some of these tests, 2) you don’t need them, 3) he’s not going to order them for you, and 4) your insurance won’t pay for them anyway, please note that Dr. Myatt will make these tests available to you here at a very reasonable cost.

Dr. Myatt’s Cardiovascular Risk Checklist Lab Tests

From Dr. Myatt’s Medical White Paper on Cardiovascular Risk Factors:

“…As many as fifty percent of all first heart attacks are last heart attacks. Half of all people who have a heart attack die from “sudden cardiac death.” People with NO conventional risk factors are more likely to die “sudden death” from a first heart attack. Sudden cardiac death is the first and only sign of heart disease in this group…”

Dr. Myatt has been able to locate and provide a comprehensive Cardiac Risk Profile that includes both currently accepted Cardiac Risk Factors and the New Emerging Cardiac Risk Factors that she discusses in her Medical White Paper.

These “emerging Risk Factors” are the ones that your conventional doctor – even your conventional cardiologist – will not test for.

Sorry – Currently unavailable – please check back soon!

ADD/ADHD in Children:

Are There Really Alternatives to Ritalin® and Prozac®?

Dana Myatt, N.M.D. and Mark Ziemann, R.N.

Attention Deficit Disorder (ADD), ADD with Hyperactivity (ADHD), and depression represent a continuum of learning and behavioral disabilities that afflict an estimated 5-10% of school-aged children. In the United States, conventional medical treatment of choice is pharmaceutical intervention.

A large body of scientific evidence suggests that these disorders are multi-factorial, representing a special challenge to the holistic physician interested in treating causes as well as symptoms. The concept of “Alternatives to Ritalin and Prozac” is, in the author’s opinion, an allopathic approach to botanical prescribing that is less efficacious than treatments which address specific etiologies. Regardless of the approach, however, botanical medicines have an important role to play in treatment.

Definition:

Definition (ADD, ADHD) Developmentally inappropriate inattention and impulsivity, with or without hyperactivity (1). The DSM-IV lists 14 signs, 8 of which must be present to make the diagnosis. They are:

Often fidgets with hands or feet and squirms in seat (restlessness),
Has difficulty remaining seated when required to do so,
Is easily distracted by external stimuli,
Has difficulty awaiting turn in games or group situations,
Often blurts out answers before questions are completed,
Has difficulty following through on instructions from others (not due to opposition but to failure of comprehension),
Has difficulty sustaining attention in tasks or play activities,
Shifts from one uncompleted task to another,
Has difficulty playing quietly,
Talks excessively,
Often interrupts or intrudes on others,
Often does not seem to listen to what is being said,
Often loses things necessary for tasks at home or at school,
Often engages in physically dangerous activities without considering consequences.

(Depression) ” . . . sad and unhappy appearance, apathy and withdrawal, reduced capacity for pleasure, feeling rejected and unloved, difficulty in sleeping, somatic complaints (headache, abdominal pain, insomnia), episodes of clowning or foolish behavior, and persistent self-blame.”(2).

Causes:

The Current Conventional “Going Line”

According to the U.S. Surgeon General and ICD-10-CM, ADHD is a metabolic form of encephalopathy which impairs the release and homeostasis of neurological chemicals, and reducing the function of the limbic system. Research indicates that the frontal lobes, their connections to the basal ganglia, and the central aspects of the cerebellum (vermis) are most likely involved in this disorder, as may be a region in the middle or medial aspect of the frontal lobe, known as the anterior cingulate.

There is increasing evidence that variants in the gene for the dopamine transporter are related to the development of ADHD [12]. This evidence is consonant with the theory of inefficacy of dopamine in people with ADD/ADHD; according to other recent studies, some people with ADHD usually have relatively high dopamine transporter levels, which clears dopamine from between neurons before the full effect is gained from dopamine. Stimulant medications used to treat ADHD are all capable of either inhibiting the action of dopamine transporter (as methylphenidate does) or promoting the release of dopamine itself (as the amphetamine-class medications do). Therefore, it is theorized that stimulant medication allows the brain to enhance the effect of dopamine by blocking dopamine transporters or increasing the release of dopamine. Currently this is the most widely accepted model of ADD/ADHD etiology in the scientific and medical community.

New studies consider the possibility that norepinephrine also plays a role. Drugs that manipulate norepinephrine levels in certain brain regions, such as atomoxetine, have shown effectiveness for managing the disorder [13] [14].

It also should be noted that despite the repeated references to the genetics of ADHD being unequivocal, according to Joseph Glenmullen, M.D., clinical instructor in psychiatry at Harvard Medical School “no claim of a gene for a psychiatric condition has stood the test of time, in spite of popular misinformation.”

The Holistic “Going Line”

ADD/ADHD can be caused by a number of factors including Nutritional deficiencies (4,5) inborn errors of metabolism (6), food allergies (7,8,9), heavy metal toxicity (10,11), malabsorption (12), prenatal influences (13,14), genetic influence (15,16), environmental (17) and cultural factors (18), yeast infection (12), food additives (7,19,20), trauma (21), and developmental factors (22).

Scope of the Problem

For ADD, ADHD: An estimated 5-10% of school-aged children are affected. Boys are 10 times more likely than girls to be diagnosed with ADD/ADHD. An estimated 3-5% of ADD/ADHD-diagnosed children will be put on Ritalin (methylphenidate). In 1995, over 6 million prescriptions were written for Americans under age 18(3).

DEPRESSION, once thought to be rare in children, has become an increasingly common diagnosis, though actual numbers are uncertain. There is a higher incidence of depression in families with a history of depressive disorder (2). Depression may occur with or without ADD/ADHD.

Diagnosis

The “cure” for ADHD relies on determining the cause. Obviously, if a nutritional deficiency is present and causative, this must be corrected. In another child, heavy metal toxicity may be the cause, in which case the treatment will be different than for those symptoms caused by nutritional imbalance.

Neurotransmitter levels should be evaluated with an NT test (urine is the specimen requirement). Heavy metal toxicity can be easily evaluated with a hair mineral analysis. Food intolerance can be screened for with a saliva test, but elimination/challenge diet is the most accurate method of determination. Yeast overgrowth in the intestinal tract can be screened with a stool test. These tests evaluate for the most common causes of ADD/ADHD. Which of them are necessary can be determined by a careful holistic physician who does an accurate history and physical exam.

Less common causes that may require evaluation include inborn errors of metabolism, trauma (especially birth trauma) and musculoskeletal imbalances.

Treatment

The “one size fits all” approach of conventional medicine has resulted in millions of US children being put on Ritalin, an amphetamine. New studies show that Ritalin can cause permanent changes in the brain and can also result in hallucinations. Clearly, if there are many different causes of this disorder, there must be a number of different treatments, each distinct and particular to the patient.

A large body of scientific evidence suggests that ADD/ADHD is multi-factorial, meaning that there is usually more than one contributing cause. This presents a special challenge to the diagnosing physician if (s)he is interested in correcting the problem and not just treating symptoms. This may also account for the large number of children placed on drug therapy, which relieves the physician and parents of the responsibility of exploring the numerous causes and contributions to ADD/ADHD. However, due to the far-reaching effects that such attention disorders and behavior problems create, many people have found that it is worthwhile to discover and correct the causes of ADD/ADHD instead of simply “dumbing down” the symptoms with drugs.

Nutritional strategies include:

Diet And Lifestyle:

Diet: Elimination/challenge to discover food allergies, then avoidance of offending foods; avoidance of artificial additives and food colorants (Feingold diet); avoidance of simple carbohydrates (sugars and refined flour products). NO stimulants: colas, chocolate, caffeine- containing foods and beverages unless indicated by an NT evaluation.

Exercise: daily. Exercise helps normalize brain chemistry.

Supplementation:

I.) Children’s Multi Vitamin: Optimal dosage according to age and body weight as listed on product label. A deficiency of any vitamin, mineral or trace mineral can lead to impaired mental performance.

II.) Omega-3 Fish Oil: A recent randomized double-blind experiment compared a fatty acid supplement with placebo in children with developmental coordination disorder (which exhibits a high degree of overlap with ADHD diagnoses). Fatty acid supplements improved spelling, reading, and behavior after three months (48). Numerous studies have shown an improvement in cognitive function, in mood, and in vision when omega 3 fatty acid supplements are given. While not directly showing a causal link between ADHD and fatty acids, increased levels of fatty acids have a beneficial effect on related behavior.

Furthermore, creating a deficiency of omega-3 fatty acids in pregnant rats produces pups that are hyperactive and that have altered brain levels of dopamine in the same brain regions as seen in humans and other rat models of hyperactivity. More research, however, is clearly needed before dietary supplements, such as those involving fatty acids can be recommended for clinical use.

III.) L-glutamine: 2,500-3,000mg per day. (This will vary depending on the age and weight of the patient).

IV.) Cal-Mag Amino: (calcium/ magnesium) [Target dose: < 10 years, 1,000mg calcium, > 10 years, 1,200- 1,500 mg calcium with corresponding dose of magnesium].

V.) Grape Seed Extract: 50 mg, 3 times per day with meals.

VI.) L-5-HTP (if indicated by an NT evaluation): dose according to weight in children. In adults, begin with 1 cap, 3 times per day and increase to 2 caps, 3 times per day after two weeks. Use only with medical supervision if antidepressant medications are also being taken.

References

1.) American Psychiatric Assoc. Diagnostic and Statistical Manual, Fourth Edition, (DSM-IV).
2.) Berkow, Robert, MD, editor-in-chief, et al. The Merck Manual of Diagnosis and Treatment, Sixteenth Edition. Merck and Company, N.J., 1992, p. 2269.
3.) John Robbins. Reclaiming our Health: Exploding the Medical Myths and Embracing the Source of True Healing. H.J. Kramer, Inc., 1996.
4.) Prinz RJ, Roberts WR, Hartman E. Dietary correlates of hyperactive behavior in children. Journal of Consulting and Clinical Psychology, 1980, 48: 760-769.
5.) Thompson HL Malnutrition as a possible factor in Learning Disabilities. Journ Learn Disab 4:27, 1971.
6.) Trattler R. Better Health Through Natural Healing. McGraw-Hill, 1988,p. 360.
7.) Feingold BF. Why Your Child is Hyperactive. New York: Random House, 1975.
8.) Trites RL, Tryphonas H, Furguson HB. Treatment of hyperactive and learning disordered children. Baltimore, Univ. Park Press, 1980.
9.) Crook WG. Can What a child eats make him dull, stupid, or hyperactive? Journ learn Disab 13:53-58, 1980.
10.) Needleman HL, Gunnoe C, Leviton A, et al. Deficits in psychological and classroom performance of children with elevated dentine lead levels. New Engl Jour of Med, 1979, 300, 13:689-695.
11.) Thatcher RW. Effects of Low Levels of Cadmium and Lead on cognitive functioning in children. Arch Environ Health 37: 159-166, 1982.
12.) Smith, L. Hyper Kids, Shaw/Spelling Assoc., Santa Monica, CA. 1990.
13.) Winick, M. Fetal Malnutrition and Growth Processes. Hospital Practice, 5:300, May 1970.
14.) Austin P, Thrash A, Thrash C. More Natural Remedies. Thrash Publications, Seale, AL, 1983, p.64.
15.) Willerman L. Activity level and hyperactivity in twins, Child Devel, 1968,27-34.
16.) Cantwaell D. Psychiatric Illness in the families of Hyperactive Children, Arch Gen Phych, 27:414, 1972.
17.) O’Leary KD, Rosenbaum A, et al.: Flourescent Lighting: A purported source of hyperactive behavior, Jour of Abnorm Child Psych, 6: 1978, p.285-289.
18.) Bandura A, Walters RH. Social Learning and Personality Development. New York: Holt, Reinhart & Winston, 1963.
19.) Rose TL. The functional relationship between artificial food colors and hyperactivity, Jour of Applied Behav Anal, 1978. P.439-446.
20.) Schoenthaler S, et al.: The impact of a low food additive and sucrose diet on Academic performance in 803 New York City public schools. Intl J of Biosoc Rec, 8:185-195, 1986, tacoma, WA.
21.) Silver L. Dr. Larry Silver’s Advice to Parents on Attention-Deficit Hyperactivity Disorder, American Psychiatric Press, Washington, D.C. 1993, p.119; p. 175-178.
22.) Dr. Myatt’s personal experience using “Patterning” and “Neurological reprogramming” techniques at the A.R.E. Clinic in Phoenix, AZ., based on the work of Drs. Doman and Delicato of the Institute for the Achievement of Human Potential in Philadelphia, PA.
23.) ChevallierA. The Encyclopedia of Medicinal Plants, D.K. Publishing, New York: 1996.
24.) O’Leary KD. Mommy, I Can’t Sit Still, New Horizon Press, 1984, p.90.
25.) Mabey R. The New Age Herbalist, Collier Books, New York:1988.
26.) Duke, J. The Green Pharmacy. Rodale Press, Emmaus, PA: 1997.
27.) Langton J. Orange-flavored Prozac to be aimed at children. Electronic Telegraph @ freepublic.com. Issue 814, August 17, 1997.
28.) Weiss RF. Herbal Medicine. AB Arcanum, Gothenburg, Sweden, 1988.
29.) Holzl J. Constituents and mechanism of action of St. John’s Wort. Zeitschr Phytother 1993;14:255-264.
30.) Ernst E. St. John’s Wort, an anti-depressant? A systematic, criteria-based review. Phytomed, 1995; 2:67-71.
31.) Vorbach EU, et al. Effectiveness and tolerance of the Hypericum extract LI 160 in comparison with imipramine: Randomized double-blind study with 135 outpatients. J Ger Psychiatry Neurol 1994; supplement 1:19-23.
32.) Werbach M, Murray M. Botanical Influences on Illness. Third Line Press, Tarzana, California, 1994, p. 31.
33.) Forester HB, et al. Planta medica, 40:4, p 309, 1980.
34.) Lutomski J, et al. Planta Medica, 27:112, 1975.
35.) Hendriks H, et al. Planta Medica, 45:150, 1982.
36.) Lindhal G, Lindwall L. Pharmacol Biochem Behav 1989;32:1065-66.
37.) Leathwood P, Chauffard F, et al. Aqueous extract of valerian root (Valerian officinalis) improves sleep quality in man. Pharmacol Biochem Behav 1982; 17:65-71.
38.) Leathwood P, Chauffard F. Aqueous extract of valerian reduces latency to fall asleep in man. Planta Medica 1985; 51:144-148.
39.) Dressing H, Riemann, D, Low M, et al. Are valerian/melissa combinations of equal value to benzodiazepine? Therapiewoche 1992; 42:726-36.
40.) Mennini T, Bernasconi P, et.al.: In vitro study on the interaction of extracts and pure compounds of valeriana officinalis roots and GABA, benzodiazepine and barbiturate receptos. Fitoterpia 1993; 64:291-300.
41.) Wohlfort R, Hansel R, Schmidt H. Planta Medica 48:120, 1983.
42.) Hoffman D. The Nervous System, Pacific NW Herbal Symposium conference notes, 1996, P. 74-85.
43.) Csupor L. Quantitative determination of kava lactones in Piper methysticum (Forster), Arch Pharm Ber Dtsch Pharm Ges 303 (3): 193-200, March 1970.
44.) Davies LP, Drew CA, Duffield P, et. al.: Kava pyrones and resins: studies on GABAA, GABAB and benzodiazepine binding sites in rodent brain. Pharmacol Toxicol 71 (2): 120-126, Aug 1992.
45.)MunteTF, Heinze HJ, et.al.: Effects of oxazepam and an extract of kava roots on event-related potential in a word recognition task. Neuropsychobiology 27 (1):46-53, 1993.
46.) Volz HP, Keiser M. Kava-kava extract WS 1490 versus placebo in anxiety disorders— a randomized, placebo controlled 25-week outpatient trial. Pharmacopsychiatry 30 91): 1-S, jan. 1997.
47.) Ruze P.Kava-induced dermopathy: a niacin deficiency? The Lancet 335 (8703):1442-1445, June 1990.
48.) Richardson and Montgomery, Pediatrics, 2005, 115:1360-1366)

Botanical Materia Medica

Group I Medications: Stimulants (Commonly used drugs in this category include methylphenidate [Ritalin], dextroamphetamine [Dexedrine], and pemoline [Cylert]). Mechanism of action: unknown, but proposed to increase the concentration of deficient norepinephrine at the nerve interface (21) or by neurotransmitter modulation at the limbic level (12).

COFFEA arabica, C. canephora – COFFEE
Coffee contains 0.06-0.32% caffeine, theobromine, theophylline and tannins. Caffeine is a strong stimulant, with bronchodilatory and diuretic effects. It’s point of action is the CNS, temporarily improving perception and motor performance (23). Some physicians have found coffee to be an acceptable alternative to psychostimulant medication, although controlled studies have shown it to be less effective then Ritalin at controlling hyperactivity (24).

COLA acuminata, C. vera, C.nitida- KOLA NUT
Cola nut contains up to 2.5% caffeine (higher than coffee), theobromine, phlobaphene, tannins, anthocyanin, proteins, fats, starch and sugars. The large seeds (nuts) have been used as a digestant and tonic for thousands of years. Kola nut stimulates the CNS (25), increases muscular strength and alertness, and counters lethargy. It has been used historically as an anti-depressant (23).

EPHEDRA sinica, E. vulgaris- EPHEDRA, MA HUANG (Chinese), MORMON TEA
Ephedra contains ephedrine, pseudoephedrine, tannins, saponins, flavones and volatile oils. The main ingredient, ephedrine, mimics adrenaline in the body and acts as a sympathetic nervous system stimulant. The synthetic alkaloid is a racemic mixture, unlike the whole herb which is levorotatory (28). This may explain why many practitioners find that the whole herb product has significant therapeutic effect at lesser dosages and with fewer side-effects than isolated ephedrine (23).

THEOBROMA cacao-COCOA
The seed pulp contains xanthines, a fixed oil, and many unidentified constituents responsible for cocoa’s characteristic flavor. The seeds are also reported to contain an endorphin-like substance that may account for chocolate’s popularity as an antidepressant substance (23).

TURNERA diffusa var. APHRODISIACA- DAMIANA
Damiana contains arbutin, volatile oils, cyanogenic glycosides,resins, gums, and a bitter amorphous principal (damianin). Damiana is best-known as an aphrodisiac and sex stimulant, although there are no reputable studies to support this claim (26). Damiana’s reputation as a CNS tonic and antidepressant is not well-substantiated, but it has been widely esteemed as a stimulant, especially when depression and anxiety occur together (23).

Other stimulant herbs to consider:

PAULLINA cupana, P. sorbilis- GUARANA
Guarana contains the same xanthine derivatives as coffee, with up to 7% caffeine, plus tannins and saponins. Indications are the same as for coffee, but long-term use is not recommended because the high tannin content can impair intestinal absorption (23).

ILEX paraguariensis-MATE
Although Mate contains xanthine derivatives in therapeutically usable amounts (0.2-1.5%), the high tannin content (16%) (23) makes it unsuitable for long-term use, especially in children.

Group II Medications: Antidepressants (Commonly used drugs in this category include tricyclics [Tofranil, Elavil, Norpramin], monoamine oxidase inhibitors (MAOI’s) [Marplan, Nardil], phenothiazines [Mellaril], and serotonin uptake inhibitors [Prozac]). A reported 70-80% of ADD/ADHD children will respond to one of the medications in category I or II or a combination of both (21) and the use of Prozac as a Group II medication is becoming increasingly common in children (27).

HYPERICUM perforatum – SAINT JOHN’S WORT
Hypericum contains a volatile oil (carophyllene), glycosides ( hypericin and pseudohypericin), flavonoids, tannins, and resins (23, 25). It occupies a special place among antidepressants of all types, being the only proven intermediary between the most powerful psychoactive drugs (Prozac, morphine, opium) and the gentle nervous system effectors such as valerian and hops (28) Various studies have found hypericum as or more effective than certain tricyclics (imipramine, maprotiline) in relieving depression (29,30, 31), with photosensitivity being the only adverse side effect (32).

Group III Medications: “Misc.”: Antipsychotics and Anti-Seizure Medications (Commonly used drugs in this category include antipsychotics [thioridazine] and anti-seizure medications [Tegretol]). Herbal analogues may have different points of action but serve as antianxiolytics and calmatives. Herbal nervines (tonics) have no equivalent in chemical medicine.

CODONOPSIS pilosula- DANG SHEN (Chinese)
Codonopsis contains triterpenoid saponins, alkaloids (perlolyrin), sterins, glycosides, polysaccharides and Tangshenoside I. Most research on the herb has been conducted in China, where it is regarded as a milder tonic and stimulant than ginseng. Codonopsis has been reported to reduce adrenaline levels (23).

MELISSA officinalis- BALM or LEMON BALM
Lemon balm contains flavonoids, triterpenes, polyphenols, tannin and up to .2% volatile oils (citral, carophyllene oxide, linalool, and citronellal). The volatile oils are thought to be the active ingredients (23). German studies have shown that the volatile oils have a CNS calmative effect (33). Balm also has carminative and antispasmodic properties, making it useful in cases where dyspepsia is part of the symptom picture (28). Although relatively safe, it should be used judiciously for long-term management due to its antithyroid effect.

PASSIFLORA incarnata- PASSION FLOWER
Passion flower contains flavonoids, cyanogenic glycosides, and maltol. Its indole alkaloid content remains in dispute (23). Passion flower is well researched as a gentle nervous system relaxant and nonaddictive sedative, although it’s mechanism of action is unknown (34).

SCUTELLARIA laterifolia- SKULLCAP, MAD-DOGWEED
Skullcap contains flavonoid glycosides (including scutellonin and scutellanein), volatile oil, bitter principals and tannins. The herb enjoys a solid reputation as a nervine, although research is sorely lacking (23,25). The Physiomedicalists (19th-century herbal practitioners) regarded skullcap as a premier herb for hysteria, epilepsy, and rabies – hence the common name “Mad-dogweed.”(23).

VALERIANA officinalis- VALERIAN
Valerian contains valpotriates, glycoside, up to 2% volatile oils (including limonene), alkaloids, choline, tannins and other constituents. Valpotriates are thought to be important in giving valerian its CNS-sedative effect (35). Numerous European studies have confirmed that valerian decreases the sleep latency period and increases quality of sleep without morning “hangover” (36,37,38) and some studies have found it to be superior to the benzodiazepine triazolam (Halcion) (39). Valerian reduces nervous activity by binding to barbiturate (GABA-A) and peripheral benzodiazepine receptors (40).

HUMULUS lupulus- HOPS
Hops contains bitter principals (lupulin,lupulon and valerianic acid), up to 1% volatile oils, flavonoids, estrogenic substances and over 100 other compounds (23,25). Hops has a proven sedative action with gastric antispasmodic effects (41).

AVENA sativa- OATS
Oats contain saponins, alkaloids, sterols, flavonoids, starch, proteins (including gluten), fats, minerals (calcium, magnesium, copper, iron, silica and zinc) and B vitamins. Oats have long been used as a nervine; the alkaloid avenine stimulates the CNS in small doses, while larger doses appear to have sedative action (28).

ESCHSCHOLZIA california- CALIFORNIA POPPY
California poppy contains flavone glycosides and alkaloids (protopine,crytopine and chelidonine), which are in the same group of isoquinolines as papverine and narcotine. The effects of poppy are considered to be nervine rather than narcotic, and its antispasmodic and analgesic effects make it useful in children for treating anxiety, nervous tension, and sleep difficulties (42).

PIPER methysticum-KAVA KAVA
Kava kava contains resins (kava lactones) and piperdine alkaloids (43). It is considered a moderately potent anxiolytic, producing stimulation followed by CNS sedation. Large doses may induce sleep. The anti-anxiety effects are similar to oxazepam (44,45), making it useful in non-psychotic anxiety with or without depression (46). Long-term use may produce “kava dermopathy,” a skin lesion of pigmented scales that occurs predominantly on the soles, palms, forehead, back and shins. The dermopathy retreats with discontinuance of the herb, and there is evidence that this reaction is due to a niacin deficiency (47).

Author’s Note:

Recommended for physicians, parents, and teachers: “Hyper Kids” by Lendon Smith, Shaw/Spelling Assoc., 1990. This workbook provides questionnaires to help physicians and parents sort out causes of ADD/ADHD – from nutrient deficiencies and allergies to malabsorbtion and yeast overgrowth. A very useful resource in differential diagnosis.

HealthBeat News

Not Just Health News… Daily Health News!

Hello All:

Do you want to keep your finger on the pulse of alternative, holistic and conventional medicine? Have I got BIG NEWS for you!

I feel privileged to have spent a week with Dr. Myatt and Nurse Mark. Yup… staying at their home, eating, sleeping (well, in a guest room!), and just plain ol’ living with them. The purpose of my visit was to gain a clearer understanding of Dr. Myatt’s needs regarding HealthBeat, but I learned a whole lot more than I bargained for.

For starters, they really do practice the things they talk about. Up at 6 AM(ish) in the morning, they each exercise (Nurse Mark goes for a run with their two dogs, Dr. M rides her bike). Then they return home and have a “Super Shake” and their before-breakfast supplements without fail. By 7am they are both at their computers, drinking freshly-brewed coffee (made with pure water and no chlorine filters — they brew this elixir in a Bodum – AKA “French Press” – that requires no paper filter). But the BIGGEST NEWS is that they are both reading the news, live on computer, even before they have breakfast and start their workday!

Dr. Myatt and Nurse Mark each have a collection of on-line newsletters that they read every morning. Dr. Myatt’s list includes JAMA (Journal of the American Medical Association), BMJ (British Medical Journal), NEJM (New England Journal of Medicine), Medline, plus holistic medical newsletters from Dr. Whittaker, David Williams, Atkins, Health Sciences Institute, Natural HealthLine, The Daily Times, CBS, NBC and a boat-load of smaller news feeds you’ve probably never heard of. Nurse mark’s list reads just as long, only with a different palette of sites. They review ALL OF THESE over their first 1-2 cups of morning coffee. If there’s something of interest, they e-mail each other with the news and discuss it. I feel comfortable in telling you that there is NO BREAKTHROUGH that has occurred overnight in any field of medicine that this medical team will not know about! Their diligence with tracking all late-breaking medical news is truly astounding.

____

[SIDEBAR: I had to laugh, maybe you will, too. While I was there, Dr. Myatt received patient and member e-mail notes (I didn’t see the notes, she just told me the “gist” of them) like the following: “Have you ever heard of glyconutrients”? and “I got this e-mail about coral calcium and I am taking it. Any good”? I’m here to tell you: if any “new nutrient” has merit, Dr. Myatt has heard about it AND reported it to you. If you haven’t heard about it from her, consider it marketing hype on the internet. I just don’t see how there could be ANYTHING of true merit or “breakthrough” status that Dr. Myatt hasn’t heard about, and probably LONG before you have, because of the multiple news feeds she monitors every morning. She also has the scientific background to sort out “marketing hype” from true science. Dr. Myatt and Nurse Mark really do this, every single morning without fail, even on weekends.]

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SO… as the new HealthBeat newsletter editor, my mind was racing….

This amazing medical duo (known locally as “The~Z~Team”) get daily updates in every field of health and medicine, but the HealthBeat newsletter comes out twice, and sometimes three times monthly. These two have a LOT more information to convey than that! So here’s what I suggested (you may send thank-you notes to me at my wellness club address!) and they went for it: a HealthBeat news “blog.” For those who don’t know, a blog is something that can be easily updated regularly if not DAILY. I set this up so Dr. Myatt can “flash” news briefs to you on a daily basis if she so chooses. I’ve made it easy and simple for her. And here’s what it means to YOU:

Dr. Myatt and Nurse Mark will continue their daily medical updates (I don’t think wild horses could stop them!). THEN, they can instantly post any important medical updates to the HealthBeat Blog. Although Dr. Myatt may be away from her computer for a day or two, you can expect (for the most part) this medical news to be updated Monday through Friday and sometimes on weekends. In other words, instead of only the bi-monthly HealthBeat, you will have access to an almost-daily HealthBeat update. Not only will you get daily breaking news, but you will have the benefit of the “new news” being screened and commented on by a physician who is an exacting scientist, thus helping you separate the “hype” from the true medical news.

TWO WAYS TO GET YOUR Daily Dose of News

Method one: If you are an RSS user, simply sign up for the newsletter and it will be “fed” to your news aggregator. If you have no idea what this means, then you are not using a service to read blogs, so you’ll use method two.

Method two: simply go to this blog link every morning.
Here it is if you need to cut and paste the link into your browser: http://radio.weblogs.com/0139131/

Once there, you will want to bookmark this link in your browser by clicking on “bookmarks.”

That’s it! Daily medical news delivered. Why not check this our right now and let me know what you think?

Jamie Jameson-White

Editor, HealthBeat Newsletter

HealthBeat News

In This Issue:

Ten Proven Reasons to take a Good Multiple Vitamin Still think taking your daily Maxi Multi or other optimal-dose vitamin/mineral supplement is a waste of time? See ten recent medical “findings” that should convince you that your multiple is very worthwhile “insurance.” PLUS, be sure to read BOGUS Science to learn about the most recent media/medical “health scam.”

Omega-3: Facts about Flax and Fish You asked for it, you got it! Find out the many benefits of Omega-3 Essential Fatty Acids from flax and fish. These are two Super-Foods you’ll definitely want to know about!

Member News and Notes
BOGUS Science Recent “health breakthroughs” that aren’t, and find out this week’s biggest, highly-publicized downright lie about nutritional supplements. A “must read” to protect yourself from bad science and “media spin.”

Upcoming topics: Keep those requests and questions coming! Future issues will detail male and female hormone replacement (natural methods), benefits of soy, and Big Fat Lies about dietary fat (this one will surprise the heck out of you!)

Ten Proven Reasons to take a Good Multiple Vitamin

While conventional medicine and newspaper headlines continue to tell us that nutritional supplementation isn’t important, the results of medical research shows just the opposite. Here are ten recent medical findings that should convince you to keep taking a high-quality, optimal potency multiple vitamin/mineral supplement. If you’re not sure what an “optimal potency” formula consists of or what you should be taking for your age and sex, refer to The Wellness Club web site’s nutritional supplementspage for an up-to-date ingredient list and optimal dose recommendations.

Harvard researchers have found that sub-optimal levels of folic acid, vitamins B6 and B12 are a risk factor for heart disease and colon and breast cancers. (Journal of the American Medical Association (JAMA) June 19, 2002)
A six-month study showing that folic acid, vitamin B12 and vitamin B6 helped prevent recurrence of blocked arteries in patients who have undergone coronary angioplasty. (Journal of the American Medical Association, August 28, 2002).
Vitamin K is a critical nutrient for skeletal integrity, with evidence of vitamin K1 supplementation reducing bone loss in healthy postmenopausal women and a significant positive relationship between vitamin K status and indices of bone health in men. (24th Annual Meeting of the American Society of Bone and Mineral Research, September 20 – 24, 2002, San Antonio, Texas)
Alzheimer’s disease: Association with zinc deficiency and cerebral vitamin B12 deficiency. (Journal of Orthol. Psychiatry (CANADA), 1984, 13/2 (97-104))
Supplementation of the elderly with vitamin E has been shown to enhance immune response, delay onset of Alzheimer’s disease, and increase resistance to oxidative injury associated with exercise. (Proc Nutr Soc. 2002;61:165-171)
Vitamin E intake, from foods or supplements, is associated with less cognitive decline with age. Arch Neurol. 2002;59:1125-1132
Researchers at Cambridge University in England looked at serum vitamin C and how long people lived. People who had the lowest levels of vitamin C were twice as likely to die compared to those with the highest serum vitamin C levels. This study was based on the findings from over 19,000 people. (Lancet 2001; 357:657-63)
26.4% of esophageal and gastric cancers are attributable to low selenium levels. (Journal of the National Cancer Institute, Mark et al., 2000)
Calcium supplementation is associated with a significant – though moderate – reduction in the risk of recurrent colorectal adenomas. The effect of calcium was independent of initial dietary fat and calcium intake. (N Engl J Med (United States) Jan 14 1999, 340 (2) p101-7.)
Data from the Nurses’ Health Study conducted at the Harvard Medical School showed that long-term supplementation with folic acid reduces the risk of colon cancer by 75% in women! 90,000 women participated in the Nurses’ Health Study, making this and especially significant finding. The authors of this study explained that folic acid obtained from supplements had a stronger protective effect against colon cancer than folic acid consumed in the diet. (Annals of Internal Medicine (1998; 129:517-524)
Regarding asthma, the lowest intakes of vitamin C and manganese (a trace mineral not to be confused with magnesium) were associated with more than five-fold increased risks of bronchial reactivity. Decreasing intakes of magnesium were also significantly associated with an increased risk of hyper-reactivity. (Thorax (United Kingdom), 1997, 52/2 (166-170)).
Antioxidant supplements reduce the risk of cataract. One study in the evaluated 410 men for 3 years to ascertain the association between serum vitamin E and the development of cortical lens opacities (cataracts). The men with the lowest level of serum vitamin E had a 3.7 times greater risk of this form of cataract compared to men with the highest serum level of vitamin E. (American Journal of Epidemiology Sept. 1996)
Encouraging moderate exercise and dietary supplementation with calcium and vitamin D… are the major nonpharmacological management measures used to prevent and treat osteoporosis. (Drugs and Aging (New Zealand), 1996, 9/6 (472-477)
Nutrient intake of patients with rheumatoid arthritis is deficient in pyridoxine (vitamin B-6), zinc, copper, and magnesium. (Journal of Rheumatology (Canada), 1996, 23/6 (990-994))

More Research Supporting Benefits of Antioxidants

By Allen S. Josephs, M.D.
President, Vitacost.com

“Recent studies further support the need for antioxidants and other nutrients to protect against cell damage, and in some cases even reduce risks of certain forms of cancer. One medical study had very promising results when participants were given a daily dose of vitamin C, vitamin E, beta carotene and selenium. Most multivitamins on the market do not qualify as good because they lack so many important antioxidants and/or use inadequate levels and forms.”

Omega-3 Fatty Acids

Also Known As: Essential Fatty Acids (EFAs), polyunsaturated fatty acids (PUFAs)

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Overview

Omega-3 fatty acids are considered essential fatty acids, which means that they are essential to human health but cannot be manufactured by the body. For this reason, omega-3 fatty acids must be obtained from food. Omega-3 fatty acids can be found in fish and certain plant oils. It is important to maintain an appropriate balance of omega-3 and omega-6 (another essential fatty acid) in the diet as these two substances work together to promote health. Also known as polyunsaturated fatty acids (PUFAs), omega-3 and omega-6 fatty acids play a crucial role in brain function as well as normal growth and development.

There are three major types of omega 3 fatty acids that are ingested in foods and used by the body: alpha-linolenic acid (ALA), eicosapentaenoic acid (EPA), and docosahexaenoic acid (DHA). Once eaten, the body converts ALA to EPA and DHA, the two types of omega-3 fatty acids more readily used by the body. Extensive research indicates that omega-3 fatty acids reduce inflammation and help prevent certain chronic diseases such as heart disease and arthritis. These essential fatty acids are highly concentrated in the brain and appear to be particularly important for cognitive and behavioral function. In fact, infants who do not get enough omega-3 fatty acids from their mothers during pregnancy are at risk for developing vision and nerve problems.

As mentioned previously, it is very important to maintain a balance between omega-3 and omega-6 fatty acids in the diet. Omega-3 fatty acids help reduce inflammation and most omega-6 fatty acids tend to promote inflammation. An inappropriate balance of these essential fatty acids contributes to the development of disease while a proper balance helps maintain and even improve health. A healthy diet should consist of roughly one to four times more omega-6 fatty acids than omega-3 fatty acids. The typical American diet tends to contain 11 to 30 times more omega-6 fatty acids than omega-3 fatty acids and many researchers believe this imbalance is a significant factor in the rising rate of inflammatory disorders in the United States.

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Uses

Studies suggest that omega-3 fatty acids may be helpful in treating a variety of conditions. The evidence is strongest for heart disease and problems that contribute to heart disease, but the range of possible uses for omega-3 fatty acids include:

High Cholesterol

Those who follow a Mediterranean-style diet tend to have higher HDL (“good”) cholesterol levels. Similar to those who follow a Mediterranean diet, Inuit Eskimos, who consume high amounts of omega-3 fatty acids from fatty fish, also tend to have increased HDL cholesterol and decreased triglycerides (fatty material that circulates in the blood). In addition, fish oil supplements containing EPA and DHA have been shown to reduce LDL (“bad”) cholesterol and triglycerides. Finally, walnuts (which are rich in ALA) have been shown to lower total cholesterol and triglycerides in people with high cholesterol.

High Blood Pressure

Several studies suggest that diets and/or supplements rich in omega-3 fatty acids lower blood pressure significantly in people with hypertension. Fish high in mercury (such as tuna) should be avoided, however, because they may increase blood pressure.

Heart Disease

One of the best ways to help prevent and treat heart disease is to eat a low-fat diet and to replace foods rich in saturated and trans-fat with those that are rich in monounsaturated and polyunsaturated fats (including omega-3 fatty acids). Evidence suggests that EPA and DHA found in fish oil help reduce risk factors for heart disease including high cholesterol and high blood pressure. There is also strong evidence that these substances can help prevent and treat atherosclerosis by inhibiting the development of plaque and blood clots, each of which tends to clog arteries. Studies of heart attack survivors have found that daily omega-3 fatty acid supplements dramatically reduce the risk of death, subsequent heart attacks, and stroke. Similarly, people who eat an ALA-rich diet are less likely to suffer a fatal heart attack.

Stroke

Strong evidence from population-based studies suggests that omega-3 fatty acid intake (primarily from fish), helps protect against stroke caused by plaque buildup and blood clots in the arteries that lead to the brain. In fact, eating at least two servings of fish per week can reduce the risk of stroke by as much as 50%. However, people who eat more than three grams of omega-3 fatty acids per day (equivalent to 3 servings of fish per day) may be at an increased risk for hemorrhagic stroke, a potentially fatal type of stroke in which an artery in the brain leaks or ruptures.

Diabetes

People with diabetes tend to have high triglyceride and low HDL levels. Omega-3 fatty acids from fish oil can help lower triglycerides and raise HDL, so people with diabetes may benefit from eating foods or taking supplements that contain DHA and EPA. ALA (from flaxseed, for example) may not have the same benefit as DHA and EPA because some people with diabetes lack the ability to efficiently convert ALA to a form of omega-3 fatty acids that the body can use readily.

Weight Loss

Many people who are overweight suffer from poor blood sugar control, diabetes, and high cholesterol. Studies suggest that overweight people who follow a weight loss program including exercise tend to achieve better control over their blood sugar and cholesterol levels when fish rich in omega-3 fatty acids (such as salmon, mackerel, and herring) is a staple in their low fat diet.

Arthritis

Most clinical studies investigating the use of omega-3 fatty acid supplements for inflammatory joint conditions have focused almost entirely on rheumatoid arthritis. Several articles reviewing the research in this area conclude that omega-3 fatty acid supplements reduce tenderness in joints, decrease morning stiffness, and allow for a reduction in the amount of medication needed for people with rheumatoid arthritis.

In addition, laboratory studies suggest that diets rich in omega-3 fatty acids (and low in omega-6 fatty acids) may benefit people with other inflammatory disorders, such as osteoarthritis. In fact, several test tube studies of cartilage-containing cells have found that omega-3 fatty acids decrease inflammation and reduce the activity of enzymes that destroy cartilage. Similarly, New Zealand green lipped mussel (Perna canaliculus), another potential source of omega-3 fatty acids, has been shown to reduce joint stiffness and pain, increase grip strength, and enhance walking pace in a small group of people with osteoarthritis. In some participants, symptoms worsened before they improved.

Osteoporosis

Studies suggest that omega-3 fatty acids such as EPA help increase levels of calcium in the body, deposit calcium in the bones, and improve bone strength. In addition, studies also suggest that people who are deficient in certain essential fatty acids (particularly EPA and gamma-linolenic acid [GLA], an omega-6 fatty acid) are more likely to suffer from bone loss than those with normal levels of these fatty acids. In a study of women over 65 with osteoporosis, those given EPA and GLA supplements experienced significantly less bone loss over three years than those who were given a placebo. Many of these women also experienced an increase in bone density.

Depression

People who do not get enough omega-3 fatty acids or do not maintain a healthy balance of omega-3 to omega-6 fatty acids in their diet may be at an increased risk for depression. The omega-3 fatty acids are important components of nerve cell membranes. They help nerve cells communicate with each other, which is an essential step in maintaining good mental health.

Levels of omega-3 fatty acids were found to be measurably low and the ratio of omega-6 to omega-3 fatty acids were particularly high in a study of patients hospitalized for depression. In a study of people with depression, those who ate a healthy diet consisting of fatty fish two to three times per week for 5 years experienced a significant reduction in feelings of depression and hostility.

Manic/Depression (Bipolar Disorder)

In a study of 30 people with bipolar disorder, those who were treated with EPA and DHA (in combination with their usual mood stabilizing medications) for four months experienced fewer mood swings and recurrence of either depression or mania than those who received placebo. A similar but larger study is currently underway at the University of California- Los Angeles School of Medicine.

Schizophrenia

Preliminary evidence suggests that people with schizophrenia experience an improvement in symptoms when given omega-3 fatty acids. However, a recent well-designed study concluded that EPA supplements are no better than placebo in improving symptoms of this condition. The conflicting results suggest that more research is needed before conclusions can be drawn about the benefit of omega-3 fatty acids for schizophrenia. Similar to diabetes, people with schizophrenia may not be able to convert ALA to EPA or DHA efficiently.

Attention Deficit/Hyperactivity Disorder (ADHD)

Children with ADHD may have low levels of certain essential fatty acids (including EPA and DHA) in their bodies. In a study of nearly 100 boys, those with lower levels of omega-3 fatty acids demonstrated more learning and behavioral problems (such as temper tantrums and sleep disturbances) than boys with normal omega-3 fatty acid levels. In animal studies, low levels of omega-3 fatty acids have been shown to lower the concentration of certain brain chemicals (such as dopamine and serotonin) related to attention and motivation. Studies that examine the ability of omega-3 supplements to improve symptoms of ADHD are still needed. At this point in time, eating foods high in omega-3 fatty acids is a reasonable approach for someone with ADHD.

Eating Disorders

Studies suggest that men and women with anorexia nervosa have lower than optimal levels of polyunsaturated fatty acids (including ALA and GLA). To prevent the complications associated with essential fatty acid deficiencies, some experts recommend that treatment programs for anorexia nervosa include PUFA-rich foods such as fish and organ meats (which include omega-6 fatty acids).

Burns

Essential fatty acids have been used to reduce inflammation and promote wound healing in burn victims. Animal research indicates that omega-3 fatty acids help promote a healthy balance of proteins in the body — protein balance is important for recovery after sustaining a burn. Further research is necessary to determine whether omega 3s benefit people in the same way.

Skin Disorders

In one study, 13 people with a particular sensitivity to the sun known as photodermatitis showed significantly less sensitivity to UV rays after taking fish oil supplements. Still, research indicates that topical sunscreens are much better at protecting the skin from damaging effects of the sun than omega-3 fatty acids. In another study of 40 people with psoriasis, those who were treated with medications and EPA supplements did better than those treated with the medications alone. In addition, many clinicians believe that flaxseed (which contains omega-3 fatty acids) is helpful for treating acne.

Inflammatory Bowel Disease (IBD)

When added to medication, such as sulfasalazine (a standard medication for IBD), omega-3 fatty acids may reduce symptoms of Crohn’s disease and ulcerative colitis — the two types of IBD. More studies to investigate this preliminary finding are under way. In animals, it appears that ALA works better at decreasing bowel inflammation than EPA and DHA. Plus, fish oil supplements can cause side effects that are similar to symptoms of IBD (such as flatulence and diarrhea). Time-release preparations may help reduce these unwanted effects.

Asthma

Preliminary research suggests that omega-3 fatty acid supplements (in the form of perilla seed oil, which is rich in ALA) may decrease inflammation and improve lung function in adults with asthma. Omega-6 fatty acids have the opposite effect: they tend to increase inflammation and worsen respiratory function. In a small, well-designed study of 29 children with asthma, those who took fish oil supplements rich in EPA and DHA for 10 months had improvement in their symptoms compared to children who took a placebo pill.

Macular Degeneration

A questionnaire administered to more than 3,000 people over the age of 49 found that those who consumed more fish in their diet were less likely to have macular degeneration (a serious age-related eye condition that can progress to blindness) than those who consumed less fish. Similarly, a study comparing 350 people with macular degeneration to 500 without found that those with a healthy dietary balance of omega-3 and omega-6 fatty acids and higher intake of fish in their diets were less likely to have this particular eye disorder. Another larger study confirms that EPA and DHA from fish, four or more times per week, may reduce the risk of developing macular degeneration. Notably, however, this same study suggests that ALA may actually increase the risk of this eye condition.

Menstrual Pain

In a study of nearly 200 Danish women, those with the highest dietary intake of omega-3 fatty acids had the mildest symptoms during menstruation.

Colon Cancer

Consuming significant amounts of foods rich in omega-3 fatty acids appears to reduce the risk of colorectal cancer. For example, Eskimos, who tend to follow a high fat diet but eat significant amounts of fish rich in omega-3 fatty acids, have a low rate of colorectal cancer. Animal studies and laboratory studies have found that omega-3 fatty acids prevent worsening of colon cancer while omega-6 fatty acids promote the growth of colon tumors. Daily consumption of EPA and DHA also appeared to slow or even reverse the progression of colon cancer in people with early stages of the disease.

However, in an animal study of rats with metastatic colon cancer (in other words, cancer that has spread to other parts of the body such as the liver), omega-3 fatty acids actually promoted the growth of cancer cells in the liver. Until more information is available, it is best for people with advanced stages of colorectal cancer to avoid omega-3 fatty acid supplements and diets rich in this substance.

Breast Cancer

Although not all experts agree, women who regularly consume foods rich in omega-3 fatty acids over many years may be less likely to develop breast cancer. In addition, the risk of dying from breast cancer may be significantly less for those who eat large quantities of omega-3 from fish and brown kelp seaweed (common in Japan). This is particularly true among women who substitute fish for meat. The balance between omega-3 and omega-6 fatty acids appears to play an important role in the development and growth of breast cancer. Further research is still needed to understand the effect that omega-3 fatty acids may have on the prevention or treatment of breast cancer. For example, several researchers speculate that omega-3 fatty acids in combination with other nutrients (namely, vitamin C, vitamin E, beta-carotene, selenium, and coenzyme Q10) may prove to be of particular value for preventing and treating breast cancer.

Prostate Cancer

Laboratory and animal studies indicate that omega-3 fatty acids (specifically, DHA and EPA) may inhibit the growth of prostate cancer. Similarly, population based studies of groups of men suggest that a low-fat diet with the addition of omega-3 fatty acids from fish or fish oil help prevent the development of prostate cancer. Like breast cancer, the balance of omega-3 to omega-6 fatty acids appears to be particularly important for reducing the risk of this condition. ALA, however, may not offer the same benefits as EPA and DHA. In fact, one recent study evaluating 67 men with prostate cancer found that they had higher levels of ALA compared to men without prostate cancer. More research in this area is needed.

Other

Although further research is needed, preliminary evidence suggests that omega-3 fatty acids may also prove helpful in protecting against certain infections and treating a variety of conditions including ulcers, migraine headaches, preterm labor, emphysema, psoriasis, glaucoma, Lyme disease, lupus, and panic attacks.

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Dietary Sources

Fish oils and plant oils are the primary dietary source of omega-3 fatty acids. Another potential source of omega-3 fatty acids is New Zealand green lipped mussels (Perna canaliculus),used for centuries by the Maories to promote good health. EPA and DHA are found in cold-water fish such as salmon, mackerel, halibut, sardines, and herring. ALA is found in flaxseeds & flaxseed oil. FISH and FLAX are the best sources. Other oils that contain significant amounts of Omega-3 are not recommended because they are also high in Omega-6. these include: canola (rapeseed) oil, soybeans, soybean oil, pumpkin seeds, pumpkin seed oil, purslane, perilla seed oil, walnuts, and walnut oil.

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Available Forms

In addition to the dietary sources described, EPA and DHA can be taken in the form of fish oil Capsules. Flaxseed, flaxseed oil, and fish oil should be kept refrigerated. Whole flaxseeds should be ground within 1 week of use to ensure maximum potency.

Be sure to buy omega-3 fatty acid supplements made by established companies who certify that their products are free of heavy metals such as mercury.

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How to Take It

Flaxseed

1 TBS. ground flax seed per day AND 1 TBS. flax oil per day OR 2 TBS. flax oil per day. (This corresponds to about 12 flax oil Capsules.

Flaxseed: 1 Tbsp two to three times per day or 2 to 4 tbsp one time per day. Grind before eating and take with lots of water.

EPA and DHA

The adequate daily intake of EPA and DHA for adults should be at least 220 mg of each per day. Two to three servings of fatty fish per week (roughly 1,250 mg EPA and DHA per day) are generally recommended to treat certain health conditions.

Fish oil supplements

3,000 to 4,000 mg standardized fish oils per day. (This amount corresponds to roughly 2 to 3 servings of fatty fish per week.)

Typically, a 1,000 mg fish oil Capsule has 180 mg EPA and 120 mg DHA

ALA

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Precautions

Because of the potential for side effects and interactions with medications, dietary supplements should be taken only under the supervision of a knowledgeable healthcare provider.

Omega-3 fatty acids should be used cautiously by people who bruise easily, have a bleeding disorder, or take blood-thinning medications because excessive amounts of omega-3 fatty acids may lead to bleeding. In fact, people who eat more than three grams of omega-3 fatty acids per day (equivalent to 3 servings of fish per day) may be at an increased risk for hemorrhagic stroke, a potentially fatal condition in which an artery in the brain leaks or ruptures.

Fish oil can cause flatulence and diarrhea. Time-release preparations may reduce these side effects, however.

People with either diabetes or schizophrenia may lack the ability to convert ALA to EPA and DHA, the forms more readily used in the body. Therefore, people with these conditions should obtain their omega-3 fatty acids from dietary sources rich in EPA and DHA.

Although studies have found that regular consumption of fish (which includes the omega-3 fatty acids EPA and DHA) may reduce the risk of macular degeneration, a recent study including two large groups of men and women found that diets rich in ALA may substantially increase the risk of this disease. More research is needed in this area. Until this information becomes available, it is best for people with macular degeneration to obtain omega-3 fatty acids from sources of EPA and DHA, rather than ALA.

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Possible Interactions

If you are currently being treated with any of the following medications, you should not use omega-3 fatty acid supplements without first talking to your healthcare provider.

Blood-thinning Medications

Omega-3 fatty acids may increase the blood-thinning effects of aspirin or warfarin. While the combination of aspirin and omega-3 fatty acids may actually be helpful under certain circumstances (such as heart disease), they should only be taken together under the guidance and supervision of your healthcare provider.

Cyclosporine

Taking omega-3 fatty acids during cyclosporine therapy may reduce toxic side effects (such as high blood pressure and kidney damage) associated with this medication in transplant patients.

Etretinate and Topical Steroids

The addition of omega-3 fatty acids (specifically EPA) to a drug regimen of etretinate and topical corticosteroids may improve symptoms of psoriasis.

Cholesterol-lowering Medications

Following certain nutritional guidelines, including increasing the amount of omega-3 fatty acids in your diet and reducing the omega-6 to omega-3 ratio, may allow a group of cholesterol lowering medications known as “statins” (such as atorvastatin, lovastatin, and simvastatin) to work more effectively.

Nonsteroidal Anti-inflammatory Drugs (NSAIDs)

In an animal study, treatment with omega-3 fatty acids reduced the risk of ulcers from nonsteroidal anti-inflammatory drugs (NSAIDs). More research is needed to evaluate whether omega-3 fatty acids would have the same effects in people.

Member News and Notes

Bogus Science

Bad advice comes in many forms and no branch of medicine is exempt from promoting faulty “science.” Just because you read something in the newspaper, the New York Times, or a health journal doesn’t necessarily make it true. Here is this month’s list of the biggest scams and shams:

1. Full-body CT scans for “general diagnosis. The CT scan is not useful for a general health-screening exam and has never been proven or even studied for this purpose. Because such scans will almost always reveal some calcifications in the body, people are led to believe that they have problems that require surgery. Let you doctor order a CT scan for specific diagnosis when indicated, but leave the “let’s just scan you for a general exam” far, far alone for now. Save your $1,000 (approximate cost of test, not covered by insurance since it isn’t proven) and take your multiple vitamin/mineral supplement instead.
2. Coral calcium: There is NO PROVEN SUPERIORITY of calcium derived from coral over other forms of calcium. Further, lead levels have been found to be unacceptably high in many samples of coral calcium.
3. Lizard spit: being touted by drug companies as a new remedy for Type II diabetes. They claim that it slightly improves glucose control in 12 weeks. Friends, type II diabetes is COMPLETELY CURABLE in 8 weeks using diet alone, but you’ll never hear that from the drug companies. Too much money can be “milked” from adult-onset diabetics who either don’t know the truth of refuse to help themselves through diet.
4. Vitamin A increases cancer risk: this is the worst of the “breakthrough news” this week. A second study using synthetic beta-carotene shows and increased risk of lung cancer in smokers. MANY population studies show a decreased risk. What’s going on? The fine print researchers forgot to report: synthetic beta-carotene does not function the same as natural beta-carotene. Natural and synthetic nutrients are not the same, that’s the only “take home message” from this study. Don’t stop taking your antioxidants on the basis of this one bit of really bad science!

CoQ10 – 100mg 60 softgel caps

CoQ10 (ubiquinone)

Super-Energizer and Potent Antioxidant

CoQ10 is a naturally-occurring antioxidant produced in the human body. It is vitally involved in energy production. CoQ10 functions as an “energizer” to mitochondria, the body’s energy producing units. Mitochondria, which produce energy the body’s “energy currency,” ATP, require CoQ10 to “spark” their production of energy units (ATP). Muscles, and the heart in particular, have high requirements for CoQ10.

CoQ10 is a potent antioxidant beneficial for:

Heart Disease (angina, arrhythmia, atherosclerosis, cardiomyopathy, heart failure, congestive heart failure, myocardial infarction (1-18)
High Blood Pressure (1,3,4,14, 53, 55)
Neurological disease (Alzheimer’s, Huntington’s, Parkinson’s) (19-37)
Immune deficiency and AIDS (40-45,52)
Periodontal disease (38-39)
Cancer (40, 44-49)
Chemotherapy side-effects (50-52)
Diabetes (53-56)
Muscular dystrophy (57-58)
fatigue / chronic fatigue / fibromyalgia (59-61)
migraine headache (62-63)
enhancing athletic performance (64-68)
male infertility (69-73)

CoQ10 is produced by the body, but age, nutrient deficiencies, disease and some medications can lower the body’s CoQ10 levels. Cholesterol-lowering drugs (statins) deplete CoQ10. (15,54,74-77)

Studies have shown that the oil-preserved form is up to 3 times better absorbed than other forms. (78-80)

Although many claims are currently made for a “new” form (ubiquinol) being “more absorbable” than ubiquinone, this has never been proven or well-studied. Learn more about this issue here:Ubiquinone (CoQ10) versus Ubiquinol: Which Is Better?

CoQ10 and it’s use in CHF (Congestive Heart Failure):

http://www.ncbi.nlm.nih.gov/pubmed/19966871
“… Coenzyme Q10 (CoQ10) is essential for electron transport within the mitochondria and hence for ATP generation and cellular energy production. We recently demonstrated that plasma levels of CoQ10 are an independent predictor of survival in a cohort of 236 patients with chronic heart failure (CHF) followed for a median of 2.69 years. This is consistent with previous studies which have shown myocardial CoQ10 depletion in CHF, and correlated with the severity of the underlying disorder. Several intervention studies have been undertaken with CoQ10 in CHF, including randomized controlled trials with mostly positive outcomes in relation to improvement in plasma levels of CoQ10. A meta-analysis showed that CoQ10 resulted in an improvement in ejection fraction of 3.7% (95%CI 1.59-5.77) and the mean increase in cardiac output was 0.28 L/minute (95%CI 0.03-0.53). In a subgroup analysis, studies with patients not taking ACE inhibitors found a 6.7% increase in ejection fraction. The ongoing Q-SYMBIO trial will address whether CoQ10 supplementation improves survival in CHF patients. CoQ10 depletion may also be a contributory factor for why statin intervention has not improved outcomes in CHF. There is an emerging evidence base in support of CoQ10 as an adjunctive therapy in CHF.”

http://faculty.washington.edu/ely/coenzq10.html
“…The majority of the clinical studies concerned the treatment of heart disease and were remarkably consistent in their conclusions: that treatment with CoQ10 significantly improved heart muscle function while producing no adverse effects or drug interactions. …”

Dr. Myatt’s Conclusion:
CoQ10 is beneficial for nearly every type of Heart Disease (angina, arrhythmia, atherosclerosis, cardiomyopathy, heart failure, congestive heart failure, myocardial infarction (1-18)

Suggested dose:

50-100 mg per day for health maintenance and anti-aging / longevity programs.

Each Capsule contains:

100 mg capsules are Wellness Club brand oil-preserved CoQ10 in easy to swallow gel Caps with Vitamin E & Beta carotene as natural preservatives

References:

1.) Adarsh K, Kaur H, Mohan V. Coenzyme Q10 (CoQ10) in isolated diastolic heart failure in hypertrophic cardiomyopathy (HCM). Biofactors. 2008;32(1-4):145-9.
2.) Berman M, Erman A, Ben-Gal T, Dvir D, Georghiou GP, Stamler A, Vered Y, Vidne BA, Aravot D. Coenzyme Q10 in patients with end-stage heart failure awaiting cardiac transplantation: a randomized, placebo-controlled study. Clin Cardiol. 2004 May;27(5):295-9.
3.) Hodgson JM, Watts GF, Playford DA, Burke V, Croft KD. Coenzyme Q10 improves blood pressure and glycaemic control: a controlled trial in subjects with type 2 diabetes. Eur J Clin Nutr. 2002 Nov;56(11):1137-42.
4.) Kumar A, Kaur H, Devi P, Mohan V. Role of Coenzyme Q10 (CoQ10) in Cardiac disease, Hypertension and Meniere- like syndrome. Pharmacol Ther. 2009 Jul 25. [Epub ahead of print]
5.) Langsjoen PH, Folkers K, Lyson K, Muratsu K, Lyson T, Langsjoen P. Pronounced increase of survival of patients with cardiomyopathy when treated with coenzyme Q10 and conventional therapy. Int J Tissue React. 1990;12(3):163-8.
6.) Langsjoen PH, Folkers K, Lyson K, Muratsu K, Lyson T, Langsjoen P. Effective and safe therapy with coenzyme Q10 for cardiomyopathy. Klin Wochenschr. 1988 Jul 1;66(13):583-90.
7.) Langsjoen P, Langsjoen A, Willis R, and Folkers K. The Aging Heart: Reversal of Diastolic Dysfunction Through the Use of Oral CoQ10 in the Elderly. Anti-Aging Medical Therapeutics. Klatz RM and Goldman R (eds.). Health Quest Publications. 1997;113-120.
8.) Langsjoen PH, Langsjoen A, Willis R, Folkers K. Treatment of hypertrophic cardiomyopathy with coenzyme Q10. Mol Aspects Med. 1997;18(S):s145-s151.
9.) Langsjoen PH, Vadhanavikit S, Folkers K. Response of patients in classes III and IV of cardiomyopathy to therapy in a blind and crossover trial with coenzyme Q10. Proc Natl Acad Sci U S A. 1985 Jun;82(12):4240-4.
10.) Mabuchi H, Higashikata T, Kawashiri M, Katsuda S, Mizuno M, Nohara A, Inazu A, Koizumi J, Kobayashi J. Reduction of serum ubiquinol-10 and ubiquinone-10 levels by atorvastatin in hypercholesterolemic patients. Journal of Atheroscler Thromb. 2005;12(2):111-9.
11.) Molyneux SL, Florkowski CM, George PM, Pilbrow AP, Frampton CM, Lever M, Richards AM. Coenzyme Q10: an independent predictor of mortality in chronic heart failure. J Am Coll Cardiol. 2008 Oct 28;52(18):1435-41.
12.) Mortensen SA. Overview on coenzyme Q10 as adjunctive therapy in chronic heart failure. Rationale, design and end-points of “Q-symbio”–a multinational trial. Biofactors. 2003;18(1-4):79-89.
13.) Mortensen S.A., Vadhanavikit S., Muratsu K., Folkers K. (1990) Coenzyme Q10: Clinical benefits with biochemical correlates suggesting a scientific breakthrough in the management of chronic heart failure. In: Int. J. Tissue React., Vol. 12 (3), pp 155-162.
14.) Rosenfeldt F, Hilton D, Pepe S, Krum H. Systematic review of effect of coenzyme Q10 in physical exercise, hypertension, and heart failure. Biofactors. 2003;18(1-4):91-100.
15.) Silver MA, Langsjoen PH, Szabo S, Patil H, Zelinger A. Effect of atorvastatin on left ventricular diastolic function and ability of coenzyme Q10 to reverse that dysfunction. Am J Cardiol. 2004 Nov 15;94(10):1306-10.
16.) Singh RB; Wander GS et al Randomized, double-blind placebo-controlled trial of coenzyme Q10 in patients with acute myocardial infarction. Cardiovasc Drugs Ther, 12(4):347-53 1998 Sep.
17.) Singh RB; Wander GS et al Cardiovasc Drugs Ther, 12(4):347-53 1998 Sep.
18.) Weant KA, Smith KM. The role of coenzyme Q10 in heart failure. Ann Pharmacother. 2005;39(9):1522-6.
19.) Beal MF. Coenzyme Q10 as a possible treatment for neurodegenerative diseases. Free Radic Res. 2002 Apr;36(4):455-60.
20.) Fallon J, Matthews RT, Hyman BT, Beal MF. MPP+ produces progressive neuronal degeneration which is mediated by oxidative stress. Exp Neurol. 1997 Mar;144(1):193-8.
21.) Feigin A, Kieburtz K, Como P, Hickey C, Claude K, Abwender D, Zimmerman C, Steinberg K, Shoulson I. Assessment of coenzyme Q10 tolerability in Huntington’s disease. Mov Disord. 1996;11(3):321-323.
22.) Ferrante KL, Shefner J, Zhang H, et al. Tolerance of high-dose (3,000 mg/day) coenzyme Q10 in ALS. Neurology. 2005 Dec 13;65(11):1834-1836.
23.) Flint Beal M, Matthews RT, Tieleman A, Shults CW. Coenzyme Q10 attenuates the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) induced loss of striatal dopamine and dopaminergic axons in aged mice. Brain Res. 1998;783:109-114.
24.) Flint Beal M, Matthews RT. Coenzyme Q10 in the central nervous system and its potential usefulness in the treatment of neurodegenerative diseases. Mol Aspects Med. 1997;18(S);s169-s179.
25.) Flint Beal M, Henshaw DR, Jenkins BG, Rosen BR, Schulz JB. Coenzyme Q10 and nicotinamide block striatal lesions produced by the mitochondrial toxin malonate. Ann Neurol. 1994;36:882-888.
26.) Jenkins BG, Brouillet E, Chen YC, Storey E, Schulz JB, Kirschner P, Beal MF, Rosen BR. Non-invasive neurochemical analysis of focal excitotoxic lesions in models of neurodegenerative illness using spectroscopic imaging. J Cereb Blood Flow Metab. 1996 May;16(3):450-61.
27.) Koroshetz WJ, Jenkins BG, Rosen BR, Flint Beal M. Energy metabolism defects in Huntington’s disease and effects of coenzyme Q10. Ann Neurol. 1997;41:160-165.
28.) Matthews RT, Yang L, Brown S, Baik MF. Coenzyme Q10 administration increases brain mitochondrial concentration and exerts neuroprotective effects. Proc Natl Acad Sci USA. 1998;95:8892-8897.
29.) Schilling G, Coonfield ML, Ross CA, Borchelt DR. Coenzyme Q10 and remacemide hydrochloride ameliorate motor deficits in a Huntington’s disease transgenic mouse model. Neurosci Lett. 2001 Nov 27;315(3):149-53.
30.) Shults CW, Oakes D, Kieburtz K, et al. Effects of Coenzyme Q10 in Early Parkinson’s Disease. Arch Neurol. 2002:59:1541-1550.
31.) Schulz B, Matthews RT, Henshaw DR, Beal MF. Neuroprotective strategies for treatment of lesions produced by mitochondrial toxins: implications for neurodegenerative diseases. Neuroscience. 1996;71(4):1043-1048.
32.) Shults CW, Flint Beal M, Fontaine D, Nakano K, Haas RH. Absorption, tolerability, and effects on mitochondrial activity of oral coenzyme Q10 in parkinsonian patients. Neurology. 1998;50:793-795.
33.) Shults CW, Haas RH, Passov D, Beal MF. Coenzyme Q10 levels correlate with the activities of complexes I and II/III in mitochondria from parkinsonian and nonparkinsonian subjects. Ann Neurol. 1997 Aug;42(2):261-4.
34.) Schulz JB, Henshaw R, Matthews RT, Flint Beal M. Coenzyme Q10 and nicotinamide and a free radical spin trap protect against MPTP neurotoxicity. Exp Neurol. 1995;132:279-283.
35.) Shults CW, Flint Beal M, Song D, Fontaine D. Pilot trial of high dosages of coenzyme Q10 in patients with Parkinson’s disease. Exp Neurol. 2004 Aug;188(2):491-4.
36.) The Huntington Study Group. A Randomized, Placebo-Controlled Trial of Coenzyme Q10 and Remacemide in Huntington’s Disease. Neurology. 2001:57:397-404.
37.) Yang L, Calingasan NY, Wille EJ, Cormier K, Smith K, Ferrante RJ, Beal MF. Combination therapy with coenzyme Q10 and creatine produces additive neuroprotective effects in models of Parkinson’s and Huntington’s diseases. J Neurochem. 2009 Jun;109(5):1427-39.
38.) Hanioka T, Tanaka M, Ojima M, Shizukuishi S, Folkers K. Effect of topical application of coenzyme Q10 on adult periodontitis. Mol Aspects Med. 1994;15 Suppl:s241-8.
39.)Nakamura R, Littarru GP, Folkers K, Wilkinson EG. Study of CoQ10-enzymes in gingiva from patients with periodontal disease and evidence for a deficiency of coenzyme Q10. Proc Natl Acad Sci U S A. 1974 Apr;71(4):1456-60.
40.) Folkers K, Hanioka T, Xia LJ, McRee JT Jr, Langsjoen P. Coenzyme Q10 increases T4/T8 ratios of lymphocytes in ordinary subjects and relevance to patients having the AIDS related complex. Biochem Biophys Res Commun. 1991 Apr 30;176(2):786-91.
41.) Folkers K, Langsjoen P, Nara Y, Muratsu K, Komorowski J, Richardson PC, Smith TH. Biochemical deficiencies of coenzyme Q10 in HIV-infection and exploratory treatment. Biochem Biophys Res Commun. 1988 Jun 16;153(2):888-96.
42.) Folkers K, Wolaniuk A. Research on coenzyme Q10 in clinical medicine and in immunomodulation. Drugs Exp Clin Res. 1985;11(8):539-45.
43.) Ravaglia G, Forti P, Maioli F, Bastagli L, Facchini A, Mariani E, Savarino L, Sassi S, Cucinotta D, Lenaz G. Effect of micronutrient status on natural killer cell immune function in healthy free-living subjects aged >/=90 y. Am J Clin Nutr. 2000 Feb;71(2):590-8.
44.) Folkers K, Morita M, McRee J Jr. The activities of coenzyme Q10 and vitamin B6 for immune responses. Biochem Biophys Res Commun. 1993 May 28;193(1):88-92.
45.) Folkers K, Brown R, Judy WV, Morita M. Biochem Biophys Res Commun. 1993 Apr 15;192(1):241-5.Survival of cancer patients on therapy with coenzyme Q10.
46.) Judy WV. Nutritional intervention in cancer prevention and treatment. American College for Advancement in Medicine Spring Conference, Ft. Lauderdale, FL. May 3, 1998.
47.) Lockwood K, Moesgaard S, Folkers K. Partial and complete regression of breast cancer in patients in relation to dosage of coenzyme Q10. Biochem Biophys Res Commun 1994;199:1504–8.
47.) Lockwood K, Moesgaard S, Hanioka T, Folkers K. Apparent partial remission of breast cancer in ‘high risk’ patients supplemented with nutritional antioxidants, essential fatty acids and coenzyme Q10. Mol Aspects Med. 1994;15 Suppl:s231-40.
48.) Lockwood K, Moesgaard S, Yamamoto T, Folkers K. Progress on therapy of breast cancer with vitamin Q10 and the regression of metastases. Biochem Biophys Res Commun 1995;212:172–7.
49.) Perumal SS, Shanthi P, Sachdanandam P. Energy-modulating vitamins–a new combinatorial therapy prevents cancer cachexia in rat mammary carcinoma.Br J Nutr. 2005 Jun;93(6):901-9.
50.) Conklin KA. Coenzyme q10 for prevention of anthracycline-induced cardiotoxicity. Integr Cancer Ther. 2005 Jun;4(2):110-30.
51.) Domae N, Sawada H, Matsuyama E, Konishi T, Uchino H. Cardiomyopathy and other chronic toxic effects induced in rabbits by doxorubicin and possible prevention by coenzyme Q10. Cancer Treat Rep. 1981 Jan-Feb;65(1-2):79-91.
52.) Folkers K, Wolaniuk A. Research on coenzyme Q10 in clinical medicine and in immunomodulation. Drugs Exp Clin Res. 1985;11(8):539-45.
53.) Chew GT, Watts GF, Davis TM, Stuckey BG, Beilin LJ, Thompson PL, Burke V, Currie PJ. Hemodynamic effects of fenofibrate and coenzyme Q10 in type 2 diabetic subjects with left ventricular diastolic dysfunction. Diabetes Care. 2008 Aug;31(8):1502-9. Epub 2008 May 16.
54.) Hamilton SJ, Chew GT, Watts GF. Coenzyme Q10 improves endothelial dysfunction in statin-treated type 2 diabetic patients. Diabetes Care. 2009 May;32(5):810-2. Epub 2009 Feb 19.
55.) Hodgson JM, Watts GF, Playford DA, Burke V, Croft KD. Coenzyme Q10 improves blood pressure and glycaemic control: a controlled trial in subjects with type 2 diabetes. Eur J Clin Nutr. 2002 Nov;56(11):1137-42.
56.) Watts GF, Playford DA, Croft KD, Ward NC, Mori TA, Burke V. Coenzyme Q(10) improves endothelial dysfunction of the brachial artery in Type II diabetes mellitus. Diabetologia. 2002 Mar;45(3):420-6.
57.) Folkers K, Simonsen R.Two successful double-blind trials with coenzyme Q10 (vitamin Q10) on muscular dystrophies and neurogenic atrophies. Biochim Biophys Acta. 1995 May 24;1271(1):281-6.
58.) Folkers K, Wolaniuk J, Simonsen R, Morishita M, Vadhanavikit S.Biochemical rationale and the cardiac response of patients with muscle disease to therapy with coenzyme Q10. Proc Natl Acad Sci U S A. 1985 Jul;82(13):4513-6.
59.) Bentler SE, Hartz AJ, Kuhn EM. Prospective observational study of treatments for unexplained chronic fatigue. J Clin Psychiatry. 2005 May;66(5):625-32.
60.) Cordero MD, Moreno-Fernández AM, deMiguel M, Bonal P, Campa F, Jiménez-Jiménez LM, Ruiz-Losada A, Sánchez-Domínguez B, Sánchez Alcázar JA, Salviati L, Navas P. Coenzyme Q10 distribution in blood is altered in patients with fibromyalgia. Clin Biochem. 2009 May;42(7-8):732-5. Epub 2008 Dec 25.
61.) Lister RE. An open, pilot study to evaluate the potential benefits of coenzyme Q10 combined with Ginkgo biloba extract in fibromyalgia syndrome. J Int Med Res. 2002 Mar-Apr;30(2):195-9.
62.) Sándor PS, Di Clemente L, Coppola G, Saenger U, Fumal A, Magis D, Seidel L, Agosti RM, Schoenen J. Efficacy of coenzyme Q10 in migraine prophylaxis: a randomized controlled trial. Neurology. 2005 Feb 22;64(4):713-5.
63.) Rozen TD, Oshinsky ML, Gebeline CA, Bradley KC, Young WB, Shechter AL, Silberstein SD. Open label trial of coenzyme Q10 as a migraine preventive. Cephalalgia. 2002 Mar;22(2):137-41.
64.) Cohen B, Gold D. Mitochondrial cytopathy in adults: What we know so far. Cleveland Clinic J Medicine 2001, 68:7,625-642.
65.) Cooke M, Iosia M, Buford T, Shelmadine B, Hudson G, Kerksick C, Rasmussen C, Greenwood M, Leutholtz B, Willoughby D, Kreider R.Effects of acute and 14-day coenzyme Q10 supplementation on exercise performance in both trained and untrained individuals. J Int Soc Sports Nutr. 2008 Mar 4;5:8.
66.) Gökbel H, Gül I, Belviranl M, Okudan N. The Effects Of Coenzyme Q10 Supplementation on Performance During Repeated Bouts of Supramaximal Exercise in Sedentary Men. J Strength Cond Res. 2009 Jul 28. [Epub ahead of print]
67.) Kon M, Tanabe K, Akimoto T, Kimura F, Tanimura Y, Shimizu K, Okamoto T, Kono I. Reducing exercise-induced muscular injury in kendo athletes with supplementation of coenzyme Q10. Br J Nutr. 2008 Feb 20;1-7. 68.) Mizuno K, Tanaka M, Nozaki S, Mizuma H, Ataka S, Tahara T, Sugino T, Shirai T, Kajimoto Y, Kuratsune H, Kajimoto O, Watanabe Y. Antifatigue effects of coenzyme Q10 during physical fatigue. Nutrition. 2008 Feb 11.
69.) Balercia G, Buldreghini E, Vignini A, Tiano L, Paggi F, Amoroso S, Ricciardo-Lamonica G, Boscaro M, Lenzi A, Littarru G. Coenzyme Q10 treatment in infertile men with idiopathic asthenozoospermia: a placebo-controlled, double-blind randomized trial. Fertil Steril. 2009 May;91(5):1785-92. 70.) Balercia G, Mancini A, Paggi F, Tiano L, Pontecorvi A, Boscaro M, Lenzi A, Littarru GP. COENZYME Q10 AND MALE INFERTILITY. J Endocrinol Invest. 2009 May 21. [Epub ahead of print]
71.) Balercia G, Mosca F, Mantero F, Boscaro M, Mancini A, Ricciardo-Lamonica G, Littarru G. Coenzyme Q(10) supplementation in infertile men with idiopathic asthenozoospermia: an open, uncontrolled pilot study. Fertil Steril. 2004 Jan;81(1):93-8.
72.) Littarru GP, Tiano L. Bioenergetic and antioxidant properties of coenzyme Q10: recent developments. Mol Biotechnol. 2007 Sep;37(1):31-7.
73.) Mancini A, De Marinis L, Littarru GP, Balercia G. An update of Coenzyme Q10 implications in male infertility: biochemical and therapeutic aspects. Biofactors. 2005;25(1-4):165-74.
74.) Joy TR, Hegele RA. Narrative review: statin-related myopathy. Ann Intern Med. 2009 Jun 16;150(12):858-68.
75.) Langsjoen PH, Langsjoen AM. The clinical use of HMG CoA-reductase inhibitors and the associated depletion of coenzyme Q10. A review of animal and human publications. Biofactors. 2003;18(1-4):101-11.
76.) Langsjoen PH, Langsjoen JO, Langsjoen AM, Lucas LA. Treatment of statin adverse effects with supplemental Coenzyme Q10 and statin drug discontinuation. Biofactors. 2005;25(1-4):147-52.
77.) Caso G, Kelly P, McNurlan MA, Lawson WE. Effect of coenzyme q10 on myopathic symptoms in patients treated with statins. Am J Cardiol. 2007 May 15;99(10):1409-12. Epub 2007 Apr 3.
78.) Bhagavan HN, Chopra RK. Plasma coenzyme Q10 response to oral ingestion of coenzyme Q10 formulations. Mitochondrion. 2007 Jun;7 Suppl:S78-88. Epub 2007 Mar 27.
79.) Chopra RK, Goldman R, Sinatra ST, Bhagavan HN. Relative bioavailability of coenzyme Q10 formulations in human subjects. Int J Vitam Nutr Res 1998;68:109–13.
80.)Weiss M, Mortensen SA, Rassig MR, et al. Bioavailability of four oral coenzyme Q10 formulations in healthy volunteers. Molec Aspects Med 1994;15:273–80.

ALZHEIMER’S DISEASE, DEMENTIA, SENILITY

Natural support for healthy mental function

An estimated 6% of the over-60 population suffer from Alzheimer’s disease, while “Senile dementia,” or non-Alzheimer’s senility, affects a similar number. The two diseases are difficult to distinguish, especially early-on. Diagnosis is a matter of clinical judgment on the part of the doctor. The only definitive diagnosis of Alzheimer’s is a post-mortem examination of the brain, where deterioration of brain cells and “scarring” are evident.

It is sometimes difficult for a lay person to distinguish “ordinary forgetfulness” from symptoms of age-related memory changes. Here are symptoms of greater concern: 1.) Memory lapses that occur more frequently and become more severe 2.) Depression, anxiety, or paranoia 3.) Loss of judgment and discrimination 4.) Mood changes: irritability, anger, loss of interest in everyday activities 5.) Loss of awareness of everyday events.

There are many non-Alzheimer’s, non-senility health problems that can cause memory and mood changes. For this reason, it is important to see your doctor for a complete physical examination. Your doctor will be able to discover if you have a health problem that is causing memory changes. Remember, most memory loss is either normal forgetfulness or caused by another illness or lifestyle factor. Secondly, and simultaneously, begin the positive steps outlined below. Simple factors such as B vitamin deficiencies can cause serious mental changes. Don’t let easily correctable memory changes happen to you!

DIET AND LIFESTYLE RECOMMENDATIONS

Eat a well-balanced diet. Lack of nutrients can cause memory changes.
Exercise regularly. Exercise improves blood flow, nutrients, and oxygen to the brain.
Avoid cigarette smoke. Cigarette smoke contains carbon monoxide, which is toxic to the brain.
“Exercise” your brain: read, work crossword puzzles, use name associations, pay attention to life!
Avoid aluminum (found in cookware, antiperspirants, antacids, beverage cans). Aluminum and other toxic metal accumulation in the brain is associated with Alzheimer’s disease.

PRIMARY SUPPORT

Take Daily Multi Vitamin and Mineral Supplement. This should include vitamins A,C,E, beta carotene, bioflavonoids, B complex vitamins (especially B1, B6, B12, folic acid), and selenium. Maxi Multi contains optimal daily doses of these nutrients.
Max EPA (fish oil): 1 cap, 3 times per day with meals to prevent or reverse inflammation. Take higher doses as directed if your hs-CRP tests are elevated. Flax oil is also beneficial but requires a biochemical conversion in the body which is deficient in many people, so fish oil is more certain.
Citicoline: A double-blind, placebo controlled study found that citicoline improved cognitive performance in Alzheimer’s patients. High-tech imaging showed that it also improved cerebral (brain) blood flow in this group of Alzheimer’s patients. According to the researchers: ” … citicoline (1,000 mg/day) is well tolerated and improves cognitive performance, cerebral blood perfusion and the brain bioelectrical activity pattern in AD [Alzheimer’s Disease] patients.” (1)

ADDITIONAL SUPPORT

Take any or all of these proven neuro-protective substances:

CoQ10: 50-300mg per day. This powerful antioxidant, produced by the body, diminishes with age. It is especially valuable for all types of heart disease. CHOLESTEROL-LOWERING DRUGS deplete CoQ10.
Turmeric: 1 capsule, 3 times per day (target dose: 900mg). Potent antioxidant, anti-inflammatory and anti-fibrin herb, turmeric acts by three different mechanisms to help protect the brain from the presumed causes of Alzheimer’s.
Ginkgo biloba: 1 cap, 2 times per day. [target dose: 240mg of a 24% flavoneglycoside formula]. Ginkgo is a potent antioxidant that also improves cerebral circulation. This herb is mentioned in The Merck Manual of (conventional) Medicine as being helpful for Alzheimer’s!
Phosphatidyl Serine: 1 cap (100mgPS), 3 times per day. PS increases brain cell communication by improving membrane fluidity.
Acetyl-L-Carnitine: 1 cap (500mg), 3 times per day between meals. A-LC acts as a powerful antioxidant in the brain.
Alpha-Lipoic Acid: 1 cap, 2-3 times per day. This neurological antioxidant chelates free iron from the forebrain, thereby protecting against free-radical induced brain aging.
Melatonin: this hormone decreases with age. It is a potent antioxidant and one of the only ones to cross the blood-brain barrier. It should be used in almost all cases of any neurological disease and is an important part of longevity and anti-aging programs.

Alzheimer’s disease and Senile Dementia are not an inevitable part of aging even though they are common in our country. Don’t let these memory-robbing diseases deprive you of YOUR Golden Years!

ADDITIONAL COMMENTS

A hair analysis should be done to rule out heavy metal and aluminum toxicity. Most conventional medical doctors do not perform this test, even though it is reliable for detecting heavy metals.
Women and men of menopausal age (40-55) should have hormone levels evaluated. A shift in the amount of sex hormones can cause memory changes.
Women of menstrual age should avoid taking ginkgo regularly. This herb has a blood-thinning effect and can cause heavier-than-normal menstrual bleeding. Consider Hypericum (St. John’s Wort) herb instead.

Related Articles:
Remembering Reagan, Avoiding Alzheimer’s

References:

1.) Alvarez XA, Mouzo R, Pichel V, Pérez P, Laredo M, Fernández-Novoa L, Corzo L, Zas R, Alcaraz M, Secades JJ, Lozano R, Cacabelos R., Methods Find Exp Clin Pharmacol. 1999 Nov;21(9):633-44. Double-blind placebo-controlled study with citicoline in APOE genotyped Alzheimer’s disease patients. Effects on cognitive performance, brain bioelectrical activity and cerebral perfusion. http://www.ncbi.nlm.nih.gov/pubmed/10669911

NeuroTonin™

Nutritional Precursors and Co-Factors to Support Optimal Broad-Spectrum Neurotransmitter Production

Neuro Tonin™ provides precursors and co-factors needed for production of all major neurotransmitters including serotonin, dopamine, epinephrine, norepinephrine, and GABA.

Adequate levels of these neurotransmitters are necessary for healthy moods, energy levels, memory, appetite control (including carbohydrate and alcohol cravings), alertness, sexual interest, normal blood pressure regulation, sound sleep, and other functions.

By helping restore normal levels of all major neurotransmitters, Neuro Tonin™ aids in creating proper balance in the brain and body needed for feelings of well-being and satisfaction.*

To learn more about feeling good by achieving healthy neurotransmitter levels, please visit our website at www.NeuroRestore.info.

*These statements have not been evaluated by the Food and Drug Administration. This product is not intended to treat, cure, or prevent any disease.

Directions: Take 4 capsules 2 times per day on an empty stomach: before breakfast and mid-afternoon (2 to 4 p.m.) is recommended. Because certain neurotransmitters increase energy, do not take after 4 p.m. Use the companion product Sero Tonin™ after 4 p.m.

Special order Only – please inquire for information.

Caution: Keep out of reach of children. Do not take while pregnant or nursing without the advise of a physician.

Storage: Keep tightly closed in a cool dry place.

Sero Tonin™

Nutritional Precursors and Co-Factors to Support Optimal Serotonin Production

Sero Tonin™ provides precursors and co-factors needed for production of the calming neurotransmitters serotonin, GABA, and melatonin.

Adequate levels of these neurotransmitters are necessary for healthy moods, appetite control (including carbohydrate and alcohol cravings), sexual interest, normal blood pressure regulation, sound sleep, and other functions.

By helping restore normal levels of calming neurotransmitters, Sero Tonin™ aids in creating proper balance in the body needed for feelings of well-being and satisfaction.*

To learn more about feeling good by achieving healthy neurotransmitter levels, please visit our website at www.NeuroRestore.info.

*These statements have not been evaluated by the Food and Drug Administration. This product is not intended to treat, cure, or prevent any disease.

Directions: Take 2 capsules on an empty stomach within one hour of bedtime or as directed by your healthcare provider.

Recommended dose is 2 capsules per day when used in conjunction with Neuro Tonin™ capsules. When used alone up to 6 capsules per day can be taken in divided doses on an empty stomach.

Special Order – Call or email to request.

Caution: Keep out of reach of children. Do not take while pregnant or nursing without the advise of a physician.

Storage: Keep tightly closed in a cool dry place.

CoQ10 – 50mg

Super-Energizer and Potent Antioxidant

CoQ10 is a potent antioxidant beneficial for:

Heart Disease (angina, arrhythmia, atherosclerosis, cardiomyopathy, heart failure, congestive heart failure, myocardial infarction (1-18)
High Blood Pressure (1,3,4,14, 53, 55)
Neurological disease (Alzheimer’s, Huntington’s, Parkinson’s) (19-37)
Immune deficiency and AIDS (40-45,52)
Periodontal disease (38-39)
Cancer (40, 44-49)
Chemotherapy side-effects (50-52)
Diabetes (53-56)
Muscular dystrophy (57-58)
fatigue / chronic fatigue / fibromyalgia (59-61)
migraine headache (62-63)
enhancing athletic performance (64-68)
male infertility (69-73)

CoQ10 is produced by the body, but age, nutrient deficiencies, disease and some medications can lower the body’s CoQ10 levels. Cholesterol-lowering drugs (statins) deplete CoQ10. (15,54,74-77)

Studies have shown that the oil-preserved form is up to 3 times better absorbed than other forms. (78-80)

Although many claims are currently made for a “new” form (ubiquinol) being “more absorbable” than ubiquinone, this has never been proven or well-studied. Learn more about this issue here: Ubiquinone (CoQ10) versus Ubiquinol: Which Is Better?

CoQ10 and it’s use in CHF (Congestive Heart Failure):

Suggested dose:

50-100 mg per day for health maintenance and anti-aging / longevity programs.

Each Capsule contains:

50 mg capsules are Wellness Club brand oil-preserved CoQ10 in easy to swallow gel Caps with Vitamin E & Beta carotene as natural preservatives.

Notes

If pregnant, nursing, or taking prescription drugs, consult your healthcare practitioner prior to use.

Manufactured by a Drug GMP audited facility.

**Based on 2000 calorie diet.

References:

CoQ10 (ubiquinone)

Super-Energizer and Potent Antioxidant

CoQ10 is a potent antioxidant beneficial for:

Heart Disease (angina, arrhythmia, atherosclerosis, cardiomyopathy, heart failure, congestive heart failure, myocardial infarction (1-18)
High Blood Pressure (1,3,4,14, 53, 55)
Neurological disease (Alzheimer’s, Huntington’s, Parkinson’s) (19-37)
Immune deficiency and AIDS (40-45,52)
Periodontal disease (38-39)
Cancer (40, 44-49)
Chemotherapy side-effects (50-52)
Diabetes (53-56)
Muscular dystrophy (57-58)
fatigue / chronic fatigue / fibromyalgia (59-61)
migraine headache (62-63)
enhancing athletic performance (64-68)
male infertility (69-73)

CoQ10 is produced by the body, but age, nutrient deficiencies, disease and some medications can lower the body’s CoQ10 levels. Cholesterol-lowering drugs (statins) deplete CoQ10. (15,54,74-77)

Studies have shown that the oil-preserved form is up to 3 times better absorbed than other forms. (78-80)

Although many claims are currently made for a “new” form (ubiquinol) being “more absorbable” than ubiquinone, this has never been proven or well-studied. Learn more about this issue here: Ubiquinone (CoQ10) versus Ubiquinol: Which Is Better?

CoQ10 and it’s use in CHF (Congestive Heart Failure):

Suggested dose:

50-100 mg per day for health maintenance and anti-aging / longevity programs.

200 to 400 mg per day for heart problems, cancer, weight loss programs and other indications.

Studies performed by the National Institutes of Health (NIH) using Vitaline ™ brand CoQ10, have used 300-400 mg (or more under medical direction) per day.

Each Capsule contains:

50 or 100 mg capsules are Wellness Club brand oil-preserved CoQ10 in easy to swallow gel Caps with Vitamin E & Beta carotene as natural preservatives.

300 and 400mg tablets are Vitaline/Integrative Therapeutics chewable wafers, the exact formulas used in the NIH Parkinson’s trials.

Product # 134 Wellness Club CoQ10 (60 softgel Caps 50 mg) 35.95

Product # 135 Wellness Club (60 softgel Caps 100 mg) 62.95

Vitaline CoQ10

Vitaline brand CoQ10 is THE CoQ10 that has been the subject of NIH studies and a recent trial showing its potential value in Parkinson’s patients.

Co10 has been shown to be potentially helpful for:

neurological health
cardiovascular health
anti-aging and longevity

NOTE: Vitaline® brand CoQ10 is available under several different labels.

Vitaline CoQ10 Vitaline Co-Enzyme Q10 Integrative Therapeutics
is the “Doctors Only” label Enzymatic Therapy
is the “Health Food Store” label Vitaline under the “Vitaline Formulas” Label is the same product

THESE ARE ALL THE EXACT SAME PRODUCT That Is Used In The NIH Studies
All are made by Vitaline and given different labels.

Product # N313 Vitaline (60 chewable wafers 300 mg With Vitamin E) 97.00

Product # N334 Vitaline (60 chewable wafers 300 mg WITHOUT Vit. E) 97.00

Product # N314 Vitaline (90 chewable wafers 400 mg With Vitamin E) 197.00

Product # N335 Vitaline (90 chewable wafers 400 mg WITHOUT Vit. E) 197.00

Vitaline, Vitamin E, and Vitamin E safety:

Some people (including some doctors) have a mistaken fear of Vitamin E, believing that “too much is dangerous.”

We have been unable to find any evidence in medical or scientific literature of any danger from taking large doses of Vitamin E. The National Institutes for Health (NIH) a US government authority places the maximum daily intake of Vitamin E at 1500 IU for healthy adults.

High intake of Vitamin E intake does tend to “thin” the blood, affecting coagulation by inhibiting platelet aggregation. People using antigoagulants or who bleed too easily may wish to discuss the use of Vitamin E with theri doctor.It has also been reported that Vitamin E in high doses may block the action of Vitamin K which is known as “the clotting factor.”

According to the label information and the Enzymatic Therapy website:

Vitaline® CoQ10 – 300 mg WITH Vitamin E contains 300 IU Vitamin E per chewable tab.

Vitaline® CoQ10 – 400 mg WITH Vitamin E contains 200 IU Vitamin E per chewable tab.

In order to achieve the daily intake of Vitamin E that you or your health care provider wish you to have you can either use a different tablet strength, either 300 or 400 mg to achieve 1200mg per day or mix your intake between Vitaline WITH and Vitaline WITHOUT Vitamin E to achieve your preferred daily intake of Vitamin E.

Vitaline 400 mg WITHOUT Vitamin E (Cherry-Vanilla flavor) Serving Size: 3 Chewable wafers Amount/Serving %DV Calories 35 Calories from fat 15 Total fat 1.5 gm 2%** Total Carbohydrate 5 gm 2%** Sugars 5 gm * Natural Coenzyme Q10 (trans-CoQ10) (ubiquinone 10) 1.2 gm *

This product does not contain

all colors used are from natural sources
artificial flavoring
corn
dairy products
gluten
ingredients of animal origin
preservatives
salt
soy
sucrose
wheat
yeast

Notes

If pregnant, nursing, or taking prescription drugs, consult your healthcare practitioner prior to use.

Manufactured by a Drug GMP audited facility.

**Based on 2000 calorie diet.

Other Ingredients

fructose, dextrose, beet juice color, silicon dioxide, natural flavors, hydrogenated vegetable oil, magnesium stearate, and malic acid.

Vitaline 400 mg WITH Vitamin E (Orange flavor) Serving Size: 3 Chewable wafers Amount/Serving %DV Calories 35 Calories from fat 15 Total fat 1.5 gm 2 %** Total Carbohydrate 5 gm 2 %** Sugars 5 gm * Natural Coenzyme Q10 (trans-CoQ10) (ubiquinone 10) 1.2 gm * Vitamin E (as dl-alpha tocopheryl acetate) 600 IU 2000 %

This product does not contain

all colors used are from natural sources
artificial flavoring
corn
dairy products
gluten
ingredients of animal origin
preservatives
salt
soy
sucrose
wheat
yeast

Notes

If pregnant, nursing, or taking prescription drugs, consult your healthcare practitioner prior to use.

Manufactured by a Drug GMP audited facility.

**Based on 2000 calorie diet.

Other Ingredients

dextrose, fructose, silicon dioxide, hydrogenated vegetable oil, natural flavor, citric acid, and magnesium stearate.

Vitaline 300 mg WITH Vitamin E (Maple Nut flavor) Serving Size: 4 Chewable wafers Amount/Serving %DV Calories 35 Calories from fat 10 Total fat 1.5 gm 2 %** Total Carbohydrate 5 gm 2 %** Sugars 5 gm * Natural Coenzyme Q10 (trans-CoQ10) (ubiquinone 10) 1.2 gm * Vitamin E (as dl-alpha tocopheryl acetate) 1200 IU 4000 %

This product does not contain

all colors used are from natural sources
artificial flavoring
corn
dairy products
gluten
ingredients of animal origin
preservatives
salt
soy
sucrose
wheat
yeast

Notes

If pregnant, nursing, or taking prescription drugs, consult your healthcare practitioner prior to use.

Manufactured by a Drug GMP audited facility.

**Based on 2000 calorie diet.

Other Ingredients

dextrose, fructose, silicon dioxide, hydrogenated vegetable oil (cottonseed), natural vanilla flavor, magnesium stearate, and natural maple nut flavor.

Vitaline 300 mg WITHOUT Vitamin E (Maple Nutflavor) Serving Size: 4 Chewable wafers Amount/Serving %DV Calories 35 Calories from fat 15 Total fat 1.5 gm 2%** Total Carbohydrate 3 gm 2%** Sugars 3 gm * Natural Coenzyme Q10 (trans-CoQ10) (ubiquinone 10) 1.2 gm *

This product does not contain

all colors used are from natural sources
artificial flavoring
corn
dairy products
gluten
ingredients of animal origin
preservatives
salt
soy
sucrose
wheat
yeast

Notes

If pregnant, nursing, or taking prescription drugs, consult your healthcare practitioner prior to use.

Manufactured by a Drug GMP audited facility.

**Based on 2000 calorie diet.

Other Ingredients

dextrose, fructose, cellulose, hydrogenated vegetable oil, partially hydrogenated soybean oil, silicon dioxide, natural vanilla flavor, magnesium stearate, sodium caseinate (milk), and natural maple nut flavor.

A Medical White Paper Presented By Dr. Dana Myatt

Dr. Myatt’s Cardiovascular Risk Checklist Lab Tests

Are There Really Alternatives to Ritalin® and Prozac®?

Dana Myatt, N.M.D. and Mark Ziemann, R.N.

Causes:

The Current Conventional “Going Line”

The Holistic “Going Line”

Scope of the Problem

Diagnosis

Treatment

Nutritional strategies include:

Diet And Lifestyle:

Supplementation:

References

Botanical Materia Medica

Other stimulant herbs to consider:

Author’s Note:

In This Issue:

CoQ10 (ubiquinone)

Super-Energizer and Potent Antioxidant

References:

Natural support for healthy mental function

Nutritional Precursors and Co-Factors to Support Optimal Broad-Spectrum Neurotransmitter Production

Nutritional Precursors and Co-Factors to Support Optimal Serotonin Production

Super-Energizer and Potent Antioxidant

Super-Energizer and Potent Antioxidant

Product # 134 Wellness Club CoQ10 (60 softgel Caps 50 mg) 35.95

Product # 135 Wellness Club (60 softgel Caps 100 mg) 62.95

Vitaline CoQ10

Vitaline brand CoQ10 is THE CoQ10 that has been the subject of NIH studies and a recent trial showing its potential value in Parkinson’s patients.

Co10 has been shown to be potentially helpful for:

NOTE: Vitaline® brand CoQ10 is available under several different labels.

Product # N313 Vitaline (60 chewable wafers 300 mg With Vitamin E) 97.00

Product # N334 Vitaline (60 chewable wafers 300 mg WITHOUT Vit. E) 97.00

Product # N314 Vitaline (90 chewable wafers 400 mg With Vitamin E) 197.00

Product # N335 Vitaline (90 chewable wafers 400 mg WITHOUT Vit. E) 197.00

References:

Learning Center

Natural Health Pharmacy

Holistic Health Handbook

What's New?

Varicose Veins

HealthBeat News

HealthBeat News